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Targeted therapy with a selective BCL-2 inhibitor in older patients with acute myeloid leukemia.
Campos, Elisabete do Vale; Pinto, Ricardo.
Afiliação
  • Campos EDV; Universidade do Porto, Faculdade de Medicina, Porto, Portugal. Electronic address: elisabete.vcampos@gmail.com.
  • Pinto R; Centro Hospitalar São João, Porto, Portugal.
Hematol Transfus Cell Ther ; 41(2): 169-177, 2019.
Article em En | MEDLINE | ID: mdl-31084767
BACKGROUND: Older patients with acute myeloid leukemia are particularly difficult to treat, as they have a high risk of comorbidities, poor performance status and less tolerability to chemotherapy, as well as a more aggressive disease biology, responsible for the resistance to treatment. There is a need to explore novel therapeutic agents that are more effective and tolerable. Venetoclax, a BCL-2 inhibitor is a promising agent, as BCL-2 overexpression is present in 84% of acute myeloid leukemia patients at diagnosis and 95% of patients at relapse and has been associated with leukemia cell survival, chemotherapy resistance and poor prognosis. OBJECTIVE: To review the available data about venetoclax in acute myeloid leukemia and how it can influence the treatment in older patients. METHODS: Using the Pubmed database, we selected 29 articles published within the last 15 years, considering preclinical and clinical trials and review studies that combined venetoclax with acute myeloid leukemia. RESULTS: Venetoclax has demonstrated promising results in preclinical and clinical trials, especially in patients with poor prognosis and the IDH mutation, with an excellent side-effect profile. However, resistance seems to develop rapidly with venetoclax monotherapy, because of antiapoptotic escape mechanisms. CONCLUSIONS: While the results with the use of venetoclax seem encouraging, it is not likely that targeting a single pathway will result in long-term disease control. The solution includes the use of combined therapy to block resistance mechanisms and enhance apoptosis, by reducing MCL-1, increasing BIM or inhibiting the complex IV in the mitochondria.
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Systematic_reviews Idioma: En Revista: Hematol Transfus Cell Ther Ano de publicação: 2019 Tipo de documento: Article País de publicação: Brasil

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Systematic_reviews Idioma: En Revista: Hematol Transfus Cell Ther Ano de publicação: 2019 Tipo de documento: Article País de publicação: Brasil