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Consequences of vascular flow on lung injury induced by mechanical ventilation.
Broccard, A F; Hotchkiss, J R; Kuwayama, N; Olson, D A; Jamal, S; Wangensteen, D O; Marini, J J.
Afiliación
  • Broccard AF; Division of Pulmonary and Critical Care Medicine, University of Minnesota, Minneapolis/St. Paul, MN, USA.
Am J Respir Crit Care Med ; 157(6 Pt 1): 1935-42, 1998 Jun.
Article en En | MEDLINE | ID: mdl-9620930
To investigate whether the magnitude of blood flow contributes to ventilator-induced lung injury, 14 sets of isolated rabbit lungs were randomized for perfusion at either 300 (Group A: n = 7) or 900 ml/ min (Group B: n = 7) while ventilated with 30 cm H2O peak static pressure. Control lungs (Group C: n = 7) were ventilated with lower peak static pressure (15 cm H2O) and perfused at 500 ml/min. Weight gain, changes in the ultrafiltration coefficient (DeltaKf) and lung static compliance (CL), and extent of hemorrhage (scored by histology) were compared. Group B had a larger decrease in CL (-13 +/- 11%) than Groups A (2 +/- 6%) and C (5 +/- 5%) (p < 0.05). Group B had more hemorrhage and gained more weight (16.2 +/- 9.5 g) than Groups A (8.7 +/- 3.4 g) and C (1.6 +/- 1.0 g) (p < 0.05 for each pairwise comparison between groups). Finally, Kf (g . min-1 . cm H2O-1 . 100 g-1) increased the most in Group B (DeltaKf = 0.26 +/- 0. 20 versus 0.17 +/- 0.10 in Group A and 0.05 +/- 0.04 in Group C; p < 0.05 for B versus C). We conclude that the intensity of lung perfusion contributes to ventilator- induced lung injury in this model.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Respiración Artificial / Síndrome de Dificultad Respiratoria / Circulación Pulmonar Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Animals Idioma: En Revista: Am J Respir Crit Care Med Asunto de la revista: TERAPIA INTENSIVA Año: 1998 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Respiración Artificial / Síndrome de Dificultad Respiratoria / Circulación Pulmonar Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Animals Idioma: En Revista: Am J Respir Crit Care Med Asunto de la revista: TERAPIA INTENSIVA Año: 1998 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos