Your browser doesn't support javascript.
loading
Upregulation of functional ryanodine receptors during in vitro aging of human diploid fibroblasts.
Huang, M S; Adebanjo, O; Moonga, B S; Goldstein, S; Lai, F A; Lipschitz, D A; Zaidi, M.
Afiliación
  • Huang MS; Department of Geriatrics, University of Arkansas for Medical Sciences, Little Rock, Arkansas, 72205, USA.
Biochem Biophys Res Commun ; 245(1): 50-2, 1998 Apr 07.
Article en En | MEDLINE | ID: mdl-9535781
We demonstrate for the first time that cellular aging in vitro is accompanied by a dramatic elevation in the levels of ryanodine receptor-bearing Ca2+ channels. These channels normally reside within microsomal membranes and gate Ca2+ release from intracellular stores. We therefore measured cytosolic Ca2+ levels in 'young' (30 mean population doublings, MPDs) and 'senescent' (53 to 58 MPDs) human diploid fibroblasts (HDFs). Application of the known ryanodine receptor modulators, caffeine or cyclic adenosine diphosphate-ribose (cADPr), triggered cytosolic Ca2+ signals in both young and senescent cells. The signal magnitude however was significantly greater in senescent compared with young HDFs. In parallel, incubation with a highly specific anti-ryanodine receptor antiserum resulted in specific immunofluorescence only in senescent HDFs. We envisage that elevated levels of functional ryanodine receptors may underlie the defective Ca2+ handling and cellular degeneration that occurs with aging.
Asunto(s)
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Regulación hacia Arriba / Senescencia Celular / Canal Liberador de Calcio Receptor de Rianodina Límite: Humans Idioma: En Revista: Biochem Biophys Res Commun Año: 1998 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Regulación hacia Arriba / Senescencia Celular / Canal Liberador de Calcio Receptor de Rianodina Límite: Humans Idioma: En Revista: Biochem Biophys Res Commun Año: 1998 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos