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CSF antigliadin antibodies and the Ramsay Hunt syndrome.
Chinnery, P F; Reading, P J; Milne, D; Gardner-Medwin, D; Turnbull, D M.
Afiliación
  • Chinnery PF; Department of Neurology, The Royal Victoria Infirmary, Newcastle upon Tyne, U.K.
Neurology ; 49(4): 1131-3, 1997 Oct.
Article en En | MEDLINE | ID: mdl-9339701
Although the association between celiac disease and progressive myoclonic ataxia is well recognized, in each of the reported cases the neurologic features began in middle adult life and usually in patients who had clinical or laboratory evidence of malabsorption. We report a case of progressive myoclonic ataxia and epilepsy (Ramsay Hunt syndrome) that began in childhood. In this patient there were no features suggestive of gluten intolerance. The presence of antigliadin antibodies in the serum and CSF suggested celiac disease was the cause of the patient's neurologic syndrome. Duodenal morphologic abnormalities reversed with treatment but no major changes were noted in the patient. Celiac disease should be considered in the differential diagnosis of myoclonic ataxia at any age, even in the absence of clinical evidence of gluten-sensitive enteropathy.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Disinergia Cerebelosa Mioclónica / Gliadina / Anticuerpos Límite: Adult / Female / Humans Idioma: En Revista: Neurology Año: 1997 Tipo del documento: Article Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Disinergia Cerebelosa Mioclónica / Gliadina / Anticuerpos Límite: Adult / Female / Humans Idioma: En Revista: Neurology Año: 1997 Tipo del documento: Article Pais de publicación: Estados Unidos