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Topotecan increases topoisomerase IIalpha levels and sensitivity to treatment with etoposide in schedule-dependent process.
Whitacre, C M; Zborowska, E; Gordon, N H; Mackay, W; Berger, N A.
Afiliación
  • Whitacre CM; Department of Medicine, Case Western Reserve University School of Medicine, Cleveland, Ohio 44106-4937, USA.
Cancer Res ; 57(8): 1425-8, 1997 Apr 15.
Article en En | MEDLINE | ID: mdl-9108439
To elucidate the effect of topoisomerase (Topo) I inhibitors in the modulation of Topo II levels and sensitivity to Topo II-directed drugs, athymic mice bearing SW480 human colon cancer xenografts were treated with simultaneous, subsequent, or distant doses of topotecan and etoposide. This in vivo study demonstrates that simultaneous administration of topotecan and etoposide results in an antagonistic response. In contrast, inhibition of Topo I by topotecan results in a compensatory increase in Topo II alpha levels associated with increasing sensitivity of tumors to subsequent treatment with the Topo II inhibitor etoposide. Furthermore, we show that Topo II alpha levels decline 5 days after the last dose of topotecan, resulting in restoration of the original response of the xenografts to etoposide. Thus, this study emphasizes the critical role of schedule dependency to optimize the effectiveness of combination chemotherapy with Topo I and Topo II inhibitors.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Protocolos de Quimioterapia Combinada Antineoplásica / ADN-Topoisomerasas de Tipo II / Isoenzimas / Proteínas de Neoplasias Tipo de estudio: Diagnostic_studies Límite: Animals / Female / Humans Idioma: En Revista: Cancer Res Año: 1997 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Protocolos de Quimioterapia Combinada Antineoplásica / ADN-Topoisomerasas de Tipo II / Isoenzimas / Proteínas de Neoplasias Tipo de estudio: Diagnostic_studies Límite: Animals / Female / Humans Idioma: En Revista: Cancer Res Año: 1997 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos