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Modulation of nicotine-induced analgesia by calcium agonist and antagonist in adult rats.
Chin, C W; Block, R C; Wu, W H; Zbuzek, V K.
Afiliación
  • Chin CW; Department of Anesthesiology, UMD-NJ Medical School, Newark 07103-2714.
Psychopharmacology (Berl) ; 110(4): 497-9, 1993.
Article en En | MEDLINE | ID: mdl-7870921
The involvement of calcium in nicotine-induced analgesia in male rats was explored using the tail-flick test. A single dose of nicotine (1 mg/kg SC) produced a maximal effect on tail-flick latency (15 s) within 8-10 min, which lasted for 4 min. Pretreatment with the calcium chelator, EDTA (250 microM/kg SC four injections at 15 min intervals), before the single dose of nicotine accelerated the onset and prolonged the duration of the nicotine-induced analgesia. The maximal effect on tail-flick latency occurred within 2 min and lasted for 10-20 min. Conversely, pretreatment with calcium chloride (1.5 mM/kg IP) attenuated nicotine-induced analgesia. It is suggested that nicotine may exert its antinociceptive effects via modulation of calcium fluxes across the neural membrane.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Agonistas de los Canales de Calcio / Bloqueadores de los Canales de Calcio / Analgesia / Nicotina Límite: Animals Idioma: En Revista: Psychopharmacology (Berl) Año: 1993 Tipo del documento: Article Pais de publicación: Alemania
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Agonistas de los Canales de Calcio / Bloqueadores de los Canales de Calcio / Analgesia / Nicotina Límite: Animals Idioma: En Revista: Psychopharmacology (Berl) Año: 1993 Tipo del documento: Article Pais de publicación: Alemania