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Molecular profiling METex14+ non-small cell lung cancer (NSCLC): Impact of histology.
Marks, Jennifer A; Gandhi, Nishant; Halmos, Balazs; Marmarelis, Melina E; Yeon Kim, So; Bazhenova, Lyudmila; Ramalingam, Suresh S; Xiu, Joanne; Walker, Phillip; Oberley, Matthew J; Ma, Patrick C; Liu, Stephen V.
Afiliación
  • Marks JA; Dana-Farber Cancer Institute, 450 Brookline Avenue, Boston, MA 02215, USA. Electronic address: jennifer_marks@dfci.harvard.edu.
  • Gandhi N; Caris Life Sciences, 4610 S 44th Pl, Phoenix, AZ 85040 USA. Electronic address: ngandhi@carisls.com.
  • Halmos B; Montefiore Medical Center, Albert Einstein Cancer Center, 1575 Blondell Ave, Bronx, NY 10461, USA. Electronic address: bahalmos@montefiore.org.
  • Marmarelis ME; University of Pennsylvania, 3400 Civic Center Boulevard West Pavilion, 2nd Floor, Philadelphia, PA 19104, USA. Electronic address: melina.marmarelis@pennmedicine.upenn.edu.
  • Yeon Kim S; Yale University, 333 Cedar St, New Haven, CT 06510, USA. Electronic address: soyeon.kim@yale.edu.
  • Bazhenova L; University of California San Diego Moores Cancer Center, 3855 Health Sciences Drive, San Diego, CA 92037, USA. Electronic address: lbazhenova@health.ucsd.edu.
  • Ramalingam SS; Winship Cancer Institute of Emory University, 1365 Clifton Rd NE Building C, Atlanta, GA 30322, USA. Electronic address: ssramal@emory.edu.
  • Xiu J; Caris Life Sciences, 4610 S 44th Pl, Phoenix, AZ 85040 USA. Electronic address: jxiu@carisls.com.
  • Walker P; Caris Life Sciences, 4610 S 44th Pl, Phoenix, AZ 85040 USA. Electronic address: pwalker@carisls.com.
  • Oberley MJ; Caris Life Sciences, 4610 S 44th Pl, Phoenix, AZ 85040 USA. Electronic address: moberley@carisls.com.
  • Ma PC; Penn State Cancer Institute, 400 University Dr, Hershey, PA 17033, USA. Electronic address: patrickma@pennstatehealth.psu.edu.
  • Liu SV; Georgetown University, 3800 Reservoir Rd NW, Washington, D.C 20007, USA. Electronic address: stephen.v.liu@gunet.georgetown.edu.
Lung Cancer ; 196: 107935, 2024 Oct.
Article en En | MEDLINE | ID: mdl-39241297
ABSTRACT

OBJECTIVES:

MET exon 14 skipping alterations (METex14+) represent a heterogeneous subgroup of non-small cell lung cancer (NSCLC) with distinct biological and genomic features. We characterized this heterogeneity in a large cohort, integrating genomic and transcriptomic profiling with clinical outcomes, to elucidate the histologic and molecular traits and survival patterns of METex14+ NSCLC. MATERIALS AND

METHODS:

NSCLC tissue samples (n = 28,739) underwent DNA-based next-generation sequencing (592 genes, NextSeq) or whole-exome sequencing (NovaSeq), RNA-sequencing including whole transcriptome sequencing (WTS, NovaSeq), and PD-L1 IHC (Dako 22C3) at Caris Life Sciences. Immune cell fractions were estimated from bulk RNA sequencing (quanTIseq). Real-world survival data (mOS) was calculated from insurance claims. Statistical analyses employed Chi-square, Fisher's exact, or Mann-Whitney U and log-rank tests and were corrected for hypothesis testing where applicable.

RESULTS:

A total of 711 METex14+ cases were detected. Of 575 cases of defined histology, 77 (13.6 %) were squamous (Sq), 474 (82.3 %) were nSq (non-squamous), and 24 (4.1 %) were adenosquamous. Mutations in POT1 and BRCA2 were enriched, and amplifications in MDM2, HMGA2, CDK4, and MET were common in METex14+ tumors. TMB-high and TP53 mutated tumors were reduced in METex14+ independent of histology. KEAP1 (2.1 vs 14.7 %) and STK11 mutations (0.8 vs 17.1 %) were reduced only in METex14+ nSq (vs METex14+ Sq, q < 0.05). While the prevalence of PD-L1 high tumors was enriched in METex14+ independent of histology, T-cell inflamed tumors were enriched only in nSq METex14+. B-cells and CD8+ T-cells (1.07-1.43-fold) were enriched in nSq METex14+, and dendritic cells (0.32 fold) were reduced only in METex14+ Sq. METex14+ tumors had a modest improvement in mOS compared to METex14- tumors (mOS = 22.9 m vs 18.6 m, HR = 0.914, p = 0.04). Moreover, METex14+ tumors who received immunotherapy (IO) had a modest improvement in survival (mOS = 27.5 m vs 21.8 m; HR = 0.803, p = 0.03) compared to those who did not receive IO. METex14+ nSq tumors were associated with improved mOS compared to METex14+ Sq tumors (mOS = 27.7 vs 8.9 m, HR = 0.493, p < 0.0001).

CONCLUSION:

METex14+ alterations are a heterogeneous subgroup of NSCLC. Our analysis reveals that METex14+ nSq exhibit improved survival compared to METex14+ Sq. The distinct genomic and transcriptomic variations across histologies warrant clinical consideration.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Carcinoma de Pulmón de Células no Pequeñas / Proteínas Proto-Oncogénicas c-met / Neoplasias Pulmonares / Mutación Límite: Aged / Female / Humans / Male / Middle aged Idioma: En Revista: Lung Cancer Asunto de la revista: NEOPLASIAS Año: 2024 Tipo del documento: Article Pais de publicación: Irlanda

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Carcinoma de Pulmón de Células no Pequeñas / Proteínas Proto-Oncogénicas c-met / Neoplasias Pulmonares / Mutación Límite: Aged / Female / Humans / Male / Middle aged Idioma: En Revista: Lung Cancer Asunto de la revista: NEOPLASIAS Año: 2024 Tipo del documento: Article Pais de publicación: Irlanda