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Gut microbiome composition and intestinal immunity in antiphospholipid syndrome patients versus healthy controls.
Jansen, Valérie Lbi; Davids, Mark; van Mourik, Dagmar Jm; Levels, Johannes Hm; Coppens, Michiel; Middeldorp, Saskia; Nieuwdorp, Max; van Mens, Thijs E.
Afiliación
  • Jansen VL; Department of (Experimental) Vascular Medicine, Amsterdam UMC Location University of Amsterdam, Amsterdam, The Netherlands.
  • Davids M; Amsterdam Cardiovascular Sciences, Pulmonary Hypertension & Thrombosis, Amsterdam, The Netherlands.
  • van Mourik DJ; Amsterdam Reproduction & Development Research Institute, Amsterdam, The Netherlands.
  • Levels JH; Department of (Experimental) Vascular Medicine, Amsterdam UMC Location University of Amsterdam, Amsterdam, The Netherlands.
  • Coppens M; Department of (Experimental) Vascular Medicine, Amsterdam UMC Location University of Amsterdam, Amsterdam, The Netherlands.
  • Middeldorp S; Amsterdam Cardiovascular Sciences, Pulmonary Hypertension & Thrombosis, Amsterdam, The Netherlands.
  • Nieuwdorp M; Department of Medicine - Thrombosis and Haemostasis, Leiden University Medical Center, Leiden, The Netherlands.
  • van Mens TE; Department of (Experimental) Vascular Medicine, Amsterdam UMC Location University of Amsterdam, Amsterdam, The Netherlands.
Lupus ; 33(12): 1373-1378, 2024 Oct.
Article en En | MEDLINE | ID: mdl-39152759
ABSTRACT

INTRODUCTION:

The gut microbiome is recognized as a factor that could potentially contribute to the persistent antibodies of antiphospholipid syndrome (APS). Gut microbial interventions can both induce and mitigate APS in mice. In human APS patients, anti-beta-2-glycoprotein I (ß2GP-1) titers correlate with antibody titers against a gut commensal protein homologous to ß2GP-1.

AIM:

To  investigate the effect of the intestinal microenvironment on human APS. Methods We cross-sectionally compared intestinal microbiota composition quantified by shotgun sequencing; fecal short chain fatty acids (SCFAs), bacterial metabolites known to affect autoimmune processes; and fecal calprotectin, an intestinal inflammatory marker, in APS patients and healthy controls.

RESULTS:

Neither alpha nor beta diversity of the gut microbiota differed between APS patients (n = 15) and controls (n = 16) and no taxa were differentially abundant. Moreover, fecal SCFAs and fecal calprotectin, did not differ between the groups.

CONCLUSION:

Gut microbiome effects on the APS phenotype are likely not driven by bacterial overabundance, SCFA production or intestinal inflammation.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Síndrome Antifosfolípido / Complejo de Antígeno L1 de Leucocito / Ácidos Grasos Volátiles / Heces / Microbioma Gastrointestinal Límite: Adult / Aged / Female / Humans / Male / Middle aged Idioma: En Revista: Lupus Asunto de la revista: REUMATOLOGIA Año: 2024 Tipo del documento: Article País de afiliación: Países Bajos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Síndrome Antifosfolípido / Complejo de Antígeno L1 de Leucocito / Ácidos Grasos Volátiles / Heces / Microbioma Gastrointestinal Límite: Adult / Aged / Female / Humans / Male / Middle aged Idioma: En Revista: Lupus Asunto de la revista: REUMATOLOGIA Año: 2024 Tipo del documento: Article País de afiliación: Países Bajos Pais de publicación: Reino Unido