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Sympathetic innervation of interscapular brown adipose tissue is not a predominant mediator of oxytocin-elicited reductions of body weight and adiposity in male diet-induced obese mice.
Edwards, Melise M; Nguyen, Ha K; Dodson, Andrew D; Herbertson, Adam J; Wolden-Hanson, Tami; Wietecha, Tomasz A; Honeycutt, Mackenzie K; Slattery, Jared D; O'Brien, Kevin D; Graham, James L; Havel, Peter J; Mundinger, Thomas O; Sikkema, Carl L; Peskind, Elaine R; Ryu, Vitaly; Taborsky, Gerald J; Blevins, James E.
Afiliación
  • Edwards MM; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Nguyen HK; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Dodson AD; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Herbertson AJ; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Wolden-Hanson T; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Wietecha TA; Division of Cardiology, Department of Medicine, University of Washington School of Medicine, Seattle, WA, United States.
  • Honeycutt MK; UW Medicine Diabetes Institute, University of Washington School of Medicine, Seattle, WA, United States.
  • Slattery JD; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • O'Brien KD; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Graham JL; Division of Cardiology, Department of Medicine, University of Washington School of Medicine, Seattle, WA, United States.
  • Havel PJ; UW Medicine Diabetes Institute, University of Washington School of Medicine, Seattle, WA, United States.
  • Mundinger TO; Department of Nutrition, University of California, Davis, Davis, CA, United States.
  • Sikkema CL; Department of Nutrition, University of California, Davis, Davis, CA, United States.
  • Peskind ER; Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, Davis, CA, United States.
  • Ryu V; Division of Metabolism, Endocrinology and Nutrition, Department of Medicine, University of Washington School of Medicine, Seattle, WA, United States.
  • Taborsky GJ; VA Puget Sound Health Care System, Office of Research and Development Medical Research Service, Department of Veterans Affairs Medical Center, Seattle, WA, United States.
  • Blevins JE; Department of Psychiatry and Behavioral Sciences, University of Washington School of Medicine, Seattle, WA, United States.
Front Endocrinol (Lausanne) ; 15: 1440070, 2024.
Article en En | MEDLINE | ID: mdl-39145314
ABSTRACT
Previous studies indicate that CNS administration of oxytocin (OT) reduces body weight in high fat diet-induced obese (DIO) rodents by reducing food intake and increasing energy expenditure (EE). We recently demonstrated that hindbrain (fourth ventricular [4V]) administration of OT elicits weight loss and elevates interscapular brown adipose tissue temperature (TIBAT, a surrogate measure of increased EE) in DIO mice. What remains unclear is whether OT-elicited weight loss requires increased sympathetic nervous system (SNS) outflow to IBAT. We hypothesized that OT-induced stimulation of SNS outflow to IBAT contributes to its ability to activate BAT and elicit weight loss in DIO mice. To test this hypothesis, we determined the effect of disrupting SNS activation of IBAT on the ability of 4V OT administration to increase TIBAT and elicit weight loss in DIO mice. We first determined whether bilateral surgical SNS denervation to IBAT was successful as noted by ≥ 60% reduction in IBAT norepinephrine (NE) content in DIO mice. NE content was selectively reduced in IBAT at 1-, 6- and 7-weeks post-denervation by 95.9 ± 2.0, 77.4 ± 12.7 and 93.6 ± 4.6% (P<0.05), respectively and was unchanged in inguinal white adipose tissue, pancreas or liver. We subsequently measured the effects of acute 4V OT (1, 5 µg ≈ 0.99, 4.96 nmol) on TIBAT in DIO mice following sham or bilateral surgical SNS denervation to IBAT. We found that the high dose of 4V OT (5 µg ≈ 4.96 nmol) elevated TIBAT similarly in sham mice as in denervated mice. We subsequently measured the effects of chronic 4V OT (16 nmol/day over 29 days) or vehicle infusions on body weight, adiposity and food intake in DIO mice following sham or bilateral surgical denervation of IBAT. Chronic 4V OT reduced body weight by 5.7 ± 2.23% and 6.6 ± 1.4% in sham and denervated mice (P<0.05), respectively, and this effect was similar between groups (P=NS). OT produced corresponding reductions in whole body fat mass (P<0.05). Together, these findings support the hypothesis that sympathetic innervation of IBAT is not necessary for OT-elicited increases in BAT thermogenesis and reductions of body weight and adiposity in male DIO mice.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Sistema Nervioso Simpático / Tejido Adiposo Pardo / Oxitocina / Adiposidad / Dieta Alta en Grasa / Ratones Endogámicos C57BL / Obesidad Límite: Animals Idioma: En Revista: Front Endocrinol (Lausanne) Año: 2024 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Sistema Nervioso Simpático / Tejido Adiposo Pardo / Oxitocina / Adiposidad / Dieta Alta en Grasa / Ratones Endogámicos C57BL / Obesidad Límite: Animals Idioma: En Revista: Front Endocrinol (Lausanne) Año: 2024 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza