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Downregulation of NAT1 Expression is Associated with Poor Prognosis and Immune Infiltration in COAD.
Xu, Houxi; Zhang, Hongqun; Sun, Songxian; Zhang, Jingyuan; Huo, Jiege; Zhou, Chunxiang.
Afiliación
  • Xu H; Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, People's Republic of China.
  • Zhang H; School of Traditional Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing People's Republic of China.
  • Sun S; Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, People's Republic of China.
  • Zhang J; The Third School of Clinical Medicine, Nanjing University of Chinese Medicine, Nanjing, People's Republic of China.
  • Huo J; School of Traditional Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing People's Republic of China.
  • Zhou C; School of Traditional Chinese Medicine, Nanjing University of Chinese Medicine, Nanjing People's Republic of China.
Pharmgenomics Pers Med ; 17: 133-148, 2024.
Article en En | MEDLINE | ID: mdl-38651072
ABSTRACT

Background:

An increasing corpus of evidence has identified the involvement of N-acetyltransferase 1 (NAT1), a member of the NAT family, in the progression of various cancers. However, the specific function of NAT1 in colon cancer (COAD) remains elusive. This study aims to decip her the role of NAT1 in COAD and its associated mechanisms.

Methods:

The Tumor Immunity Evaluation Resource (TIMER), The Cancer Genome Atlas (TCGA), and the Gene Expression Omnibus (GEO) databases were employed to assess the NAT1 expression level in COAD. The differential expression between COAD and normal colon tissue was further validated using quantitative real-time reverse-transcription PCR (RT-qPCR) and Western blot (WB) analyses. Additionally, survival analysis of NAT1 in COAD was carried out using the PrognoScan database and TCGA dataset. The functions of NAT1 were explored through gene set enrichment analysis (GSEA) and immuno-infiltration analysis.

Results:

There was a significant reduction in NAT1 expression in COAD samples compared to normal tissue. Notably, low NAT1 expression in COAD correlated significantly with various clinical parameters such as tumor stage (T stage, N stage, M stage, pathologic stage), primary therapy outcome, carcinoembryonic antigen (CEA) level, and lymphatic invasion. The downregulation of NAT1 was also strongly linked with poor outcomes in overall survival (OS), progression-free interval (PFI), and disease-specific survival (DSS). Cox regression analysis highlighted NAT1 as an independent prognostic indicator for overall survival in COAD patients. GSEA results revealed NAT1's involvement in multiple pathways, including the neuroactive ligand-receptor interaction, olfactory transduction, olfactory signaling, extracellular matrix receptor interaction, calcium signaling, and focal adhesion pathways. Furthermore, NAT1 expression was found to significantly correlate with infiltration levels of various immune cells.

Conclusion:

The findings reveal NAT1's potential as a valuable prognostic biomarker for COAD. Moreover, its associated mechanisms offer insights that might pave the way for therapeutic interventions for COAD patients.
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Pharmgenomics Pers Med Año: 2024 Tipo del documento: Article Pais de publicación: Nueva Zelanda

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Pharmgenomics Pers Med Año: 2024 Tipo del documento: Article Pais de publicación: Nueva Zelanda