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A phosphodiesterase 4 (PDE4) inhibitor, amlexanox, reduces neuroinflammation and neuronal death after pilocarpine-induced seizure.
Yang, Hyun Wook; Kho, A Ra; Lee, Song Hee; Kang, Beom Seok; Park, Min Kyu; Lee, Chang Jun; Park, Se Wan; Woo, Seo Young; Kim, Dong Yeon; Jung, Hyun Ho; Choi, Bo Young; Yang, Won Il; Song, Hong Ki; Choi, Hui Chul; Park, Jin Kyu; Suh, Sang Won.
Afiliación
  • Yang HW; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: akqjqtj5@hallym.ac.kr.
  • Kho AR; Neuroregeneration and Stem Cell Programs, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA; Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Electronic address: akho3@jhu.edu.
  • Lee SH; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: sshlee@hallym.ac.kr.
  • Kang BS; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: bskang@hallym.ac.kr.
  • Park MK; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: D22029@hallym.ac.kr.
  • Lee CJ; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: doog0716@hallym.ac.kr.
  • Park SW; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: M2022087@hallym.ac.kr.
  • Woo SY; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: M22091@hallym.ac.kr.
  • Kim DY; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: M22525@hallym.ac.kr.
  • Jung HH; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: wjdgusgh1021@hallym.ac.kr.
  • Choi BY; Department of Physical Education, Hallym University, Chuncheon 24252, Republic of Korea; Institute of Sport Science, Hallym University, Chuncheon 24252, Republic of Korea. Electronic address: bychoi@hallym.ac.kr.
  • Yang WI; Institute of Sport Science, Hallym University, Chuncheon 24252, Republic of Korea; Department of Sport Industry Studies, Yonsei University, Seoul 03722, Republic of Korea. Electronic address: wonil4u@hallym.ac.kr.
  • Song HK; Neurology, Kangdong Sacred Heart Hospital, Seoul 05355, Republic of Korea; Hallym Institute of Epilepsy Research, Hallym University, Chuncheon 24252, Republic of Korea. Electronic address: hksong0@hallym.oc.kr.
  • Choi HC; Neurology, Hallym University Chuncheon Sacred Heart Hospital, Chuncheon 24253, Republic of Korea; Hallym Institute of Epilepsy Research, Hallym University, Chuncheon 24252, Republic of Korea. Electronic address: dohchi@hallym.ac.kr.
  • Park JK; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea. Electronic address: jinkyupark71@hallym.ac.kr.
  • Suh SW; Department of Physiology, Neurology, Hallym University, College of Medicine, 1-Okcheon Dong, 39 Hallymdaehak-gil, Chuncheon 200-708, Republic of Korea; Hallym Institute of Epilepsy Research, Hallym University, Chuncheon 24252, Republic of Korea. Electronic address: swsuh@hallym.ac.kr.
Neurotherapeutics ; 21(4): e00357, 2024 Jul.
Article en En | MEDLINE | ID: mdl-38631990
ABSTRACT
Epilepsy, a complex neurological disorder, is characterized by recurrent seizures caused by aberrant electrical activity in the brain. Central to this study is the role of lysosomal dysfunction in epilepsy, which can lead to the accumulation of toxic substrates and impaired autophagy in neurons. Our focus is on phosphodiesterase-4 (PDE4), an enzyme that plays a crucial role in regulating intracellular cyclic adenosine monophosphate (cAMP) levels by converting it into adenosine monophosphate (AMP). In pathological states, including epilepsy, increased PDE4 activity contributes to a decrease in cAMP levels, which may exacerbate neuroinflammatory responses. We hypothesized that amlexanox, an anti-inflammatory drug and non-selective PDE4 inhibitor, could offer neuroprotection by addressing lysosomal dysfunction and mitigating neuroinflammation, ultimately preventing neuronal death in epileptic conditions. Our research utilized a pilocarpine-induced epilepsy animal model to investigate amlexanox's potential benefits. Administered intraperitoneally at a dose of 100 â€‹mg/kg daily following the onset of a seizure, we monitored its effects on lysosomal function, inflammation, neuronal death, and cognitive performance in the brain. Tissue samples from various brain regions were collected at predetermined intervals for a comprehensive analysis. The study's results were significant. Amlexanox effectively improved lysosomal function, which we attribute to the modulation of zinc's influx into the lysosomes, subsequently enhancing autophagic processes and decreasing the release of inflammatory factors. Notably, this led to the attenuation of neuronal death in the hippocampal region. Additionally, cognitive function, assessed through the modified neurological severity score (mNSS) and the Barnes maze test, showed substantial improvements after treatment with amlexanox. These promising outcomes indicate that amlexanox has potential as a therapeutic agent in the treatment of epilepsy and related brain disorders. Its ability to combat lysosomal dysfunction and neuroinflammation positions it as a potential neuroprotective intervention. While these findings are encouraging, further research and clinical trials are essential to fully explore and validate the therapeutic efficacy of amlexanox in epilepsy management.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Pilocarpina / Convulsiones / Muerte Celular / Inhibidores de Fosfodiesterasa 4 / Enfermedades Neuroinflamatorias / Aminopiridinas / Neuronas Límite: Animals Idioma: En Revista: Neurotherapeutics Asunto de la revista: NEUROLOGIA Año: 2024 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Pilocarpina / Convulsiones / Muerte Celular / Inhibidores de Fosfodiesterasa 4 / Enfermedades Neuroinflamatorias / Aminopiridinas / Neuronas Límite: Animals Idioma: En Revista: Neurotherapeutics Asunto de la revista: NEUROLOGIA Año: 2024 Tipo del documento: Article Pais de publicación: Estados Unidos