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Bidirectional relation between dipeptidyl peptidase 4 and angiotensin II type I receptor signaling.
Martins, Flavia L; Ribeiro-Silva, Joao Carlos; Nistala, Ravi; Girardi, Adriana C C.
Afiliación
  • Martins FL; Laboratory of Genetics and Molecular Cardiology, Heart Institute (InCor), University of Sao Paulo Medical School, Sao Paulo, Brazil.
  • Ribeiro-Silva JC; Division of Nephrology, Department of Medicine, University of Missouri School of Medicine, Columbia, Missouri, United States.
  • Nistala R; Department of Ophthalmology & Visual Sciences, State University of New York Upstate Medical University, Syracuse, New York, United States.
  • Girardi ACC; Division of Nephrology, Department of Medicine, University of Missouri School of Medicine, Columbia, Missouri, United States.
Am J Physiol Cell Physiol ; 326(4): C1203-C1211, 2024 04 01.
Article en En | MEDLINE | ID: mdl-38581656
ABSTRACT
Cardiometabolic diseases are often associated with heightened levels of angiotensin II (Ang II), which accounts for the observed oxidative stress, inflammation, and fibrosis. Accumulating evidence indicates a parallel upregulation of dipeptidyl dipeptidase 4 (DPP4) activity in cardiometabolic diseases, with its inhibition shown to mitigate oxidative stress, inflammation, and fibrosis. These findings highlight an overlap between the pathophysiological mechanisms used by Ang II and DPP4. Recent evidence demonstrates that targeted inhibition of DPP4 prevents the rise in Ang II and its associated molecules in experimental models of cardiometabolic diseases. Similarly, inhibitors of the angiotensin I-converting enzyme (ACE) or Ang II type 1 receptor (AT1R) blockers downregulate DPP4 activity, establishing a bidirectional relationship between DPP4 and Ang II. Here, we discuss the current evidence supporting the cross talk between Ang II and DPP4, along with the potential mechanisms promoting this cross regulation. A comprehensive analysis of this bidirectional relationship across tissues will advance our understanding of how DPP4 and Ang II collectively promote the development and progression of cardiometabolic diseases.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Angiotensina II / Enfermedades Cardiovasculares Límite: Humans Idioma: En Revista: Am J Physiol Cell Physiol Asunto de la revista: FISIOLOGIA Año: 2024 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Angiotensina II / Enfermedades Cardiovasculares Límite: Humans Idioma: En Revista: Am J Physiol Cell Physiol Asunto de la revista: FISIOLOGIA Año: 2024 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Estados Unidos