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Dosage-dependent effects of FGFR2W290R mutation on craniofacial shape and cellular dynamics of the basicranial synchondroses.
Richbourg, Heather A; Vidal-García, Marta; Brakora, Katherine A; Devine, Jay; Takenaka, Risa; Young, Nathan M; Gong, Siew-Ging; Neves, Amanda; Hallgrímsson, Benedikt; Marcucio, Ralph S.
Afiliación
  • Richbourg HA; Department of Orthopedic Surgery, University of California, San Francisco, San Francisco, California, USA.
  • Vidal-García M; Alberta Children's Hospital Research Institute, University of Calgary, Calgary, Alberta, Canada.
  • Brakora KA; The McCaig Institute for Bone and Joint Health, University of Calgary, Calgary, Alberta, Canada.
  • Devine J; Department of Cell Biology and Anatomy, Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada.
  • Takenaka R; Department of Neuroscience and Experimental Therapeutics, Texas A&M University School of Medicine, Bryan, Texas, USA.
  • Young NM; Alberta Children's Hospital Research Institute, University of Calgary, Calgary, Alberta, Canada.
  • Gong SG; The McCaig Institute for Bone and Joint Health, University of Calgary, Calgary, Alberta, Canada.
  • Neves A; Department of Cell Biology and Anatomy, Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada.
  • Hallgrímsson B; Department of Orthopedic Surgery, University of California, San Francisco, San Francisco, California, USA.
  • Marcucio RS; Molecular and Cellular Biology, University of Washington, Seattle, Washington, USA.
Anat Rec (Hoboken) ; 2024 Feb 26.
Article en En | MEDLINE | ID: mdl-38409943
ABSTRACT
Craniosynostosis is a common yet complex birth defect, characterized by premature fusion of the cranial sutures that can be syndromic or nonsyndromic. With over 180 syndromic associations, reaching genetic diagnoses and understanding variations in underlying cellular mechanisms remains a challenge. Variants of FGFR2 are highly associated with craniosynostosis and warrant further investigation. Using the missense mutation FGFR2W290R , an effective mouse model of Crouzon syndrome, craniofacial features were analyzed using geometric morphometrics across developmental time (E10.5-adulthood, n = 665 total). Given the interrelationship between the cranial vault and basicranium in craniosynostosis patients, the basicranium and synchondroses were analyzed in perinates. Embryonic time points showed minimal significant shape differences. However, hetero- and homozygous mutant perinates and adults showed significant differences in shape and size of the cranial vault, face, and basicranium, which were associated with cranial doming and shortening of the basicranium and skull. Although there were also significant shape and size differences associated with the basicranial bones and clear reductions in basicranial ossification in cleared whole-mount samples, there were no significant alterations in chondrocyte cell shape, size, or orientation along the spheno-occipital synchondrosis. Finally, shape differences in the cranial vault and basicranium were interrelated at perinatal stages. These results point toward the possibility that facial shape phenotypes in craniosynostosis may result in part from pleiotropic effects of the causative mutations rather than only from the secondary consequences of the sutural defects, indicating a novel direction of research that may shed light on the etiology of the broad changes in craniofacial morphology observed in craniosynostosis syndromes.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Anat Rec (Hoboken) Asunto de la revista: ANATOMIA Año: 2024 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Anat Rec (Hoboken) Asunto de la revista: ANATOMIA Año: 2024 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos