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Therapeutic-like activity of cannabidiolic acid methyl ester in the MK-801 mouse model of schizophrenia: Role for cannabinoid CB1 and serotonin-1A receptors.
Richardson, Brandon; Clarke, Courtney; Blundell, Jacqueline; Bambico, Francis R.
Afiliación
  • Richardson B; Memorial University of Newfoundland and Labrador, St. John's, Newfoundland, Canada.
  • Clarke C; Memorial University of Newfoundland and Labrador, St. John's, Newfoundland, Canada.
  • Blundell J; Memorial University of Newfoundland and Labrador, St. John's, Newfoundland, Canada.
  • Bambico FR; Memorial University of Newfoundland and Labrador, St. John's, Newfoundland, Canada.
Eur J Neurosci ; 59(9): 2403-2415, 2024 May.
Article en En | MEDLINE | ID: mdl-38385841
ABSTRACT
Schizophrenia is a psychotic disorder with an increasing prevalence and incidence over the last two decades. The condition presents with a diverse array of positive, negative, and cognitive impairments. Conventional treatments often yield unsatisfactory outcomes, especially with negative symptoms. We investigated the role of prefrontocortical (PFC) N-methyl-D-aspartate receptors (NMDARs) in the pathophysiology and development of schizophrenia. We explored the potential therapeutic effects of cannabidiolic acid (CBDA) methyl ester (HU-580), an analogue of CBDA known to act as an agonist of the serotonin-1A receptor (5-HT1AR) and an antagonist of cannabinoid type 1 receptor (CB1R). C57BL/6 mice were intraperitoneally administered the NMDAR antagonist, dizocilpine (MK-801, .3 mg/kg) once daily for 17 days. After 7 days, they were concurrently given HU-580 (.01 or .05 µg/kg) for 10 days. Behavioural deficits were assessed at two time points. We conducted enzyme-linked immunosorbent assays to measure the concentration of PFC 5-HT1AR and CB1R. We found that MK-801 effectively induced schizophrenia-related behaviours including hyperactivity, social withdrawal, increased forced swim immobility, and cognitive deficits. We discovered that low-dose HU-580 (.01 µg/kg), but not the high dose (.05 µg/kg), attenuated hyperactivity, forced swim immobility and cognitive deficits, particularly in female mice. Our results revealed that MK-801 downregulated both CB1R and 5-HT1AR, an effect that was blocked by both low- and high-dose HU-580. This study sheds light on the potential antipsychotic properties of HU-580, particularly in the context of NMDAR-induced dysfunction. Our findings could contribute significantly to our understanding of schizophrenia pathophysiology and offer a promising avenue for exploring the therapeutic potential of HU-580 and related compounds in alleviating symptoms.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Esquizofrenia / Maleato de Dizocilpina / Receptor Cannabinoide CB1 / Receptor de Serotonina 5-HT1A / Modelos Animales de Enfermedad / Ratones Endogámicos C57BL Límite: Animals Idioma: En Revista: Eur J Neurosci Asunto de la revista: NEUROLOGIA Año: 2024 Tipo del documento: Article País de afiliación: Canadá Pais de publicación: Francia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Esquizofrenia / Maleato de Dizocilpina / Receptor Cannabinoide CB1 / Receptor de Serotonina 5-HT1A / Modelos Animales de Enfermedad / Ratones Endogámicos C57BL Límite: Animals Idioma: En Revista: Eur J Neurosci Asunto de la revista: NEUROLOGIA Año: 2024 Tipo del documento: Article País de afiliación: Canadá Pais de publicación: Francia