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Adipose tissue-derived exosomes alleviate particulate matter-induced inflammatory response and skin barrier damage in atopic dermatitis-like triple-cell model.
Roh, Yoon Jin; Choi, Yong Hee; Shin, Sun Hye; Lee, Mi-Kyung; Won, Yu Jin; Lee, Jun Ho; Cho, Byong Seung; Park, Kui Young; Seo, Seong Jun.
Afiliación
  • Roh YJ; Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea.
  • Choi YH; Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea.
  • Shin SH; Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea.
  • Lee MK; Department of Laboratory Medicine, Chung-Ang University College of Medicine, Seoul, Korea.
  • Won YJ; ExoCoBio Exosome Institute (EEI), ExoCoBio Inc., Seoul, Korea.
  • Lee JH; ExoCoBio Exosome Institute (EEI), ExoCoBio Inc., Seoul, Korea.
  • Cho BS; ExoCoBio Exosome Institute (EEI), ExoCoBio Inc., Seoul, Korea.
  • Park KY; Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea.
  • Seo SJ; Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea.
PLoS One ; 19(1): e0292050, 2024.
Article en En | MEDLINE | ID: mdl-38241278
ABSTRACT
Recently, particulate matter (PM) has been shown to exacerbate atopic dermatitis (AD) by inducing an inflammatory response. Meanwhile, several studies revealed that exosomes derived from adipose tissue-derived mesenchymal stem cells promote wound healing and alleviate inflammation via their regenerative and immunomodulatory capacities. Our study aimed to investigate the effects of human adipose tissue-derived mesenchymal stem cell-derived (ASC)-exosomes in PM-induced AD. An AD-like triple-cell model was established by treating human keratinocytes, dermal fibroblasts, and mast cells with polyinosinicpolycytidylic acid (Poly IC) and interleukin 1 alpha (IL-1α). The effects of PM and ASC-exosomes on the expression of pro-inflammatory cytokines and skin barrier proteins were examined using quantitative real-time polymerase chain reaction, western blotting, and immunofluorescence. PM increased pro-inflammatory cytokines (IL-6, IL-1ß, and IL-1α) and decreased the anti-inflammatory cytokine IL-10, while the mRNA expression of skin barrier proteins (loricrin and filaggrin) decreased. However, when the cells were treated with ASC-exosomes, the PM-induced effects on pro-inflammatory cytokines and skin barrier proteins were reversed. Our results confirmed that PM-induced inflammation and skin barrier damage were alleviated by ASC-exosomes in our AD-like triple-cell model. These data suggest that ASC-exosomes can serve as a therapeutic agent for PM-exacerbated AD.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Dermatitis Atópica / Exosomas Límite: Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2024 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Dermatitis Atópica / Exosomas Límite: Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2024 Tipo del documento: Article Pais de publicación: Estados Unidos