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Genetic Characterization of Intimin Gene (eae) in Clinical Shiga Toxin-Producing Escherichia coli Strains from Pediatric Patients in Finland.
Wang, Lei; Bai, Xiangning; Ylinen, Elisa; Zhang, Ji; Saxén, Harri; Matussek, Andreas.
Afiliación
  • Wang L; Department of Microbiology, Division of Laboratory Medicine, Oslo University Hospital and University of Oslo, 0372 Oslo, Norway.
  • Bai X; Jinan Center for Disease Control and Prevention, Jinan 250021, China.
  • Ylinen E; Department of Microbiology, Division of Laboratory Medicine, Oslo University Hospital and University of Oslo, 0372 Oslo, Norway.
  • Zhang J; Department of Clinical Microbiology, Division of Laboratory Medicine, Karolinska Institutet, 141 52 Stockholm, Sweden.
  • Saxén H; Department of Pediatric Nephrology and Transplantation, New Children's Hospital, University of Helsinki and Helsinki University Hospital, 00029 Helsinki, Finland.
  • Matussek A; Fonterra Research and Development Centre, Dairy Farm Road, Palmerston North 4442, New Zealand.
Toxins (Basel) ; 15(12)2023 11 23.
Article en En | MEDLINE | ID: mdl-38133173
ABSTRACT
Shiga toxin (Stx)-producing Escherichia coli (STEC) infections cause outbreaks of severe disease in children ranging from bloody diarrhea to hemolytic uremic syndrome (HUS). The adherent factor intimin, encoded by eae, can facilitate the colonization process of strains and is frequently associated with severe disease. The purpose of this study was to examine and analyze the prevalence and polymorphisms of eae in clinical STEC strains from pediatric patients under 17 years old with and without HUS, and to assess the pathogenic risk of different eae subtypes. We studied 240 STEC strains isolated from pediatric patients in Finland with whole genome sequencing. The gene eae was present in 209 (87.1%) strains, among which 49 (23.4%) were from patients with HUS, and 160 (76.6%) were from patients without HUS. O157H7 (126, 60.3%) was the most predominant serotype among eae-positive STEC strains. Twenty-three different eae genotypes were identified, which were categorized into five eae subtypes, i.e., γ1, ß3, ε1, θ and ζ3. The subtype eae-γ1 was significantly overrepresented in strains from patients aged 5-17 years, while ß3 and ε1 were more commonly found in strains from patients under 5 years. All O157H7 strains carried eae-γ1; among non-O157 strains, strains of each serotype harbored one eae subtype. No association was observed between the presence of eae/its subtypes and HUS. However, the combination of eae-γ1+stx2a was significantly associated with HUS. In conclusion, this study demonstrated a high occurrence and genetic variety of eae in clinical STEC from pediatric patients under 17 years old in Finland, and that eae is not essential for STEC-associated HUS. However, the combination of certain eae subtypes with stx subtypes, i.e., eae-γ1+stx2a, may be used as risk predictors for the development of severe disease in children.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Adhesinas Bacterianas / Proteínas de Escherichia coli / Infecciones por Escherichia coli / Escherichia coli Shiga-Toxigénica / Síndrome Hemolítico-Urémico Límite: Adolescent / Child / Humans País/Región como asunto: Europa Idioma: En Revista: Toxins (Basel) Año: 2023 Tipo del documento: Article País de afiliación: Noruega Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Adhesinas Bacterianas / Proteínas de Escherichia coli / Infecciones por Escherichia coli / Escherichia coli Shiga-Toxigénica / Síndrome Hemolítico-Urémico Límite: Adolescent / Child / Humans País/Región como asunto: Europa Idioma: En Revista: Toxins (Basel) Año: 2023 Tipo del documento: Article País de afiliación: Noruega Pais de publicación: Suiza