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Divergence in aerobic capacity and energy expenditure influence metabolic tissue mitochondrial protein synthesis rates in aged rats.
Franczak, Edziu; Maurer, Adrianna; Drummond, Vivien Csikos; Kugler, Benjamin A; Wells, Emily; Wenger, Madi; Peelor, Frederick F; Crosswhite, Abby; McCoin, Colin S; Koch, Lauren G; Britton, Steven L; Miller, Benjamin F; Thyfault, John P.
Afiliación
  • Franczak E; Department of Cell Biology and Physiology, Medical Center, The University of Kansas, Kansas City, KS, 66160, USA.
  • Maurer A; Kansas City Veterans Affairs Medical Center, Kansas City, MO, 64128, USA.
  • Drummond VC; Department of Cell Biology and Physiology, Medical Center, The University of Kansas, Kansas City, KS, 66160, USA.
  • Kugler BA; Department of Cell Biology and Physiology, Medical Center, The University of Kansas, Kansas City, KS, 66160, USA.
  • Wells E; Department of Cell Biology and Physiology, Medical Center, The University of Kansas, Kansas City, KS, 66160, USA.
  • Wenger M; Kansas Center for Metabolism and Obesity Research, Kansas City, MO, 64128, USA.
  • Peelor FF; KU Diabetes Institute and Department of Internal Medicine-Division of Endocrinology and Metabolism, The University of Kansas Medical Center, 3901 Rainbow Boulevard, Hemenway Life Sciences Innovation Center, Mailstop 3043, Kansas City, KS, 66160, USA.
  • Crosswhite A; Department of Cell Biology and Physiology, Medical Center, The University of Kansas, Kansas City, KS, 66160, USA.
  • McCoin CS; Department of Cell Biology and Physiology, Medical Center, The University of Kansas, Kansas City, KS, 66160, USA.
  • Koch LG; Kansas Center for Metabolism and Obesity Research, Kansas City, MO, 64128, USA.
  • Britton SL; KU Diabetes Institute and Department of Internal Medicine-Division of Endocrinology and Metabolism, The University of Kansas Medical Center, 3901 Rainbow Boulevard, Hemenway Life Sciences Innovation Center, Mailstop 3043, Kansas City, KS, 66160, USA.
  • Miller BF; Oklahoma Medical Research Foundation, Oklahoma City, OK, 73104, USA.
  • Thyfault JP; Oklahoma Medical Research Foundation, Oklahoma City, OK, 73104, USA.
Geroscience ; 46(2): 2207-2222, 2024 Apr.
Article en En | MEDLINE | ID: mdl-37880490
Age-associated declines in aerobic capacity promote the development of various metabolic diseases. In rats selectively bred for high/low intrinsic aerobic capacity, greater aerobic capacity reduces susceptibility to metabolic disease while increasing longevity. However, little remains known how intrinsic aerobic capacity protects against metabolic disease, particularly with aging. Here, we tested the effects of aging and intrinsic aerobic capacity on systemic energy expenditure, metabolic flexibility and mitochondrial protein synthesis rates using 24-month-old low-capacity (LCR) or high-capacity runner (HCR) rats. Rats were fed low-fat diet (LFD) or high-fat diet (HFD) for eight weeks, with energy expenditure (EE) and metabolic flexibility assessed utilizing indirect calorimetry during a 48 h fast/re-feeding metabolic challenge. Deuterium oxide (D2O) labeling was used to assess mitochondrial protein fraction synthesis rates (FSR) over a 7-day period. HCR rats possessed greater EE during the metabolic challenge. Interestingly, HFD induced changes in respiratory exchange ratio (RER) in male and female rats, while HCR female rat RER was largely unaffected by diet. In addition, analysis of protein FSR in skeletal muscle, brain, and liver mitochondria showed tissue-specific adaptations between HCR and LCR rats. While brain and liver protein FSR were altered by aerobic capacity and diet, these effects were less apparent in skeletal muscle. Overall, we provide evidence that greater aerobic capacity promotes elevated EE in an aged state, while also regulating metabolic flexibility in a sex-dependent manner. Modulation of mitochondrial protein FSR by aerobic capacity is tissue-specific with aging, likely due to differential energetic requirements by each tissue.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Metabolismo Energético / Enfermedades Metabólicas Límite: Animals Idioma: En Revista: Geroscience Año: 2024 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Metabolismo Energético / Enfermedades Metabólicas Límite: Animals Idioma: En Revista: Geroscience Año: 2024 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza