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Identification of Genomic Regions Implicated in Susceptibility to Schistosoma mansoni Infection in a Murine Backcross Genetic Model.
Hernández-Goenaga, Juan; López-Abán, Julio; Blanco-Gómez, Adrián; Vicente, Belén; Burguillo, Francisco Javier; Pérez-Losada, Jesús; Muro, Antonio.
Afiliación
  • Hernández-Goenaga J; Grupo de Enfermedades Infecciosas y Tropicales (e-INTRO), IBSAL-CIETUS (Instituto de Investigación Biomédica de Salamanca, Centro de Investigación de Enfermedades Tropica-les de la Universidad de Salamanca), Facultad de Farmacia, Universidad de Salamanca, Ldo. Méndez Nieto s/n, 37007 Salamanca, Spai
  • López-Abán J; Grupo de Enfermedades Infecciosas y Tropicales (e-INTRO), IBSAL-CIETUS (Instituto de Investigación Biomédica de Salamanca, Centro de Investigación de Enfermedades Tropica-les de la Universidad de Salamanca), Facultad de Farmacia, Universidad de Salamanca, Ldo. Méndez Nieto s/n, 37007 Salamanca, Spai
  • Blanco-Gómez A; Instituto de Investigación Biomédica de Salamanca (IBSAL), Complejo Asistencial Universitario de Salamanca, Hospital Virgen de la Vega, 37007 Salamanca, Spain.
  • Vicente B; Instituto de Biología Molecular del Cáncer (IBMCC), Centro de Investigación del Cáncer (CIC)-CSIC, Laboratory 20, 37007 Salamanca, Spain.
  • Burguillo FJ; Grupo de Enfermedades Infecciosas y Tropicales (e-INTRO), IBSAL-CIETUS (Instituto de Investigación Biomédica de Salamanca, Centro de Investigación de Enfermedades Tropica-les de la Universidad de Salamanca), Facultad de Farmacia, Universidad de Salamanca, Ldo. Méndez Nieto s/n, 37007 Salamanca, Spai
  • Pérez-Losada J; Departamento de Química-Física, Facultad de Farmacia, Universidad de Salamanca, C/Donantes de Sangre s/n. Campus Unamuno, 37007 Salamanca, Spain.
  • Muro A; Instituto de Investigación Biomédica de Salamanca (IBSAL), Complejo Asistencial Universitario de Salamanca, Hospital Virgen de la Vega, 37007 Salamanca, Spain.
Int J Mol Sci ; 24(19)2023 Sep 30.
Article en En | MEDLINE | ID: mdl-37834216
Only a small number of infected people are highly susceptible to schistosomiasis, showing high levels of infection or severe liver fibrosis. The susceptibility to schistosome infection is influenced by genetic background. To assess the genetic basis of susceptibility and identify the chromosomal regions involved, a backcross strategy was employed to generate high variation in schistosomiasis susceptibility. This strategy involved crossing the resistant C57BL/6J mouse strain with the susceptible CBA/2J strain. The resulting F1 females (C57BL/6J × CBA/2J) were then backcrossed with CBA/2J males to generate the backcross (BX) cohort. The BX mice exhibited a range of phenotypes, with disease severity varying from mild to severe disease, lacking a fully resistant group. We observed four levels of infection intensity using cluster and principal component analyses and K-means based on parasitological, pathological, and immunological trait measurements. The mice were genotyped with 961 informative SNPs, leading to the identification of 19 new quantitative trait loci (QTL) associated with parasite burden, liver lesions, white blood cell populations, and antibody responses. Two QTLs located on chromosomes 15 and 18 were linked to the number of granulomas, liver lesions, and IgM levels. The corresponding syntenic human regions are located in chromosomes 8 and 18. None of the significant QTLs had been reported previously.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Esquistosomiasis / Esquistosomiasis mansoni / Neoplasias Hepáticas Límite: Animals / Female / Humans / Male Idioma: En Revista: Int J Mol Sci Año: 2023 Tipo del documento: Article Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Esquistosomiasis / Esquistosomiasis mansoni / Neoplasias Hepáticas Límite: Animals / Female / Humans / Male Idioma: En Revista: Int J Mol Sci Año: 2023 Tipo del documento: Article Pais de publicación: Suiza