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Impaired parasympathetic function in long-COVID postural orthostatic tachycardia syndrome - a case-control study.
Rigo, Stefano; Urechie, Vasile; Diedrich, Andrè; Okamoto, Luis E; Biaggioni, Italo; Shibao, Cyndya A.
Afiliación
  • Rigo S; Department of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, TN, USA. s.rigo96@gmail.com.
  • Urechie V; Department of Biomedical Sciences, Humanitas University, Via Rita Levi Montalcini 4, Pieve Emanuele, Milan, 20090, Italy. s.rigo96@gmail.com.
  • Diedrich A; Department of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, TN, USA.
  • Okamoto LE; Department of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, TN, USA.
  • Biaggioni I; Department of Biomedical Engineering, Vanderbilt University, Nashville, TN, USA.
  • Shibao CA; Department of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, TN, USA.
Bioelectron Med ; 9(1): 19, 2023 Sep 06.
Article en En | MEDLINE | ID: mdl-37670400
PURPOSE: Eighty percent of patients infected by SARS-CoV-2 report persistence of one symptom beyond the 4-week convalescent period. Those with orthostatic tachycardia and orthostatic symptoms mimicking postural tachycardia syndrome, they are defined as Long-COVID POTS [LCP]. This case-control study investigated potential differences in autonomic cardiovascular regulation between LCP patients and healthy controls. METHODS: Thirteen LCP and 16 healthy controls, all female subjects, were studied without medications. Continuous blood pressure and ECG were recorded during orthostatic stress test, respiratory sinus arrhythmia, and Valsalva maneuver. Time domain and power spectral analysis of heart rate [HR] and systolic blood pressure [SBP] variability were computed characterizing cardiac autonomic control and sympathetic peripheral vasoconstriction. RESULTS: LCP had higher deltaHR (+ 40 ± 6 vs. + 21 ± 3 bpm, p = 0.004) and deltaSBP (+ 8 ± 4 vs. -1 ± 2 mmHg, p = 0.04) upon standing; 47% had impaired Valsalva maneuver ratio compared with 6.2% in controls (p = 0.01). Spectral analysis revealed that LCP had lower RMSSD (32.1 ± 4.6 vs. 48.9 ± 6.8 ms, p = 0.04) and HFRRI, both in absolute (349 ± 105 vs. 851 ± 253ms2, p = 0.03) and normalized units (32 ± 4 vs. 46 ± 4 n.u., p = 0.02). LFSBP was similar between groups. CONCLUSIONS: LCP have reduced cardiovagal modulation, but normal sympathetic cardiac and vasoconstrictive functions. Impaired parasympathetic function may contribute to the pathogenesis of Long-COVID POTS syndrome.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Observational_studies Idioma: En Revista: Bioelectron Med Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Observational_studies Idioma: En Revista: Bioelectron Med Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido