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Regulation of Presynaptic Release Machinery by Cell Adhesion Molecules.
Uchigashima, Motokazu; Hayashi, Yasunori; Futai, Kensuke.
Afiliación
  • Uchigashima M; Department of Cellular Neuropathology, Brain Research Institute, Niigata University, Niigata, Japan.
  • Hayashi Y; Department of Pharmacology, Kyoto University Graduate School of Medicine, Kyoto, Japan.
  • Futai K; Brudnick Neuropsychiatric Research Institute, Department of Neurobiology, University of Massachusetts Chan Medical School, Worcester, MA, USA. kensuke.futai@umassmed.edu.
Adv Neurobiol ; 33: 333-356, 2023.
Article en En | MEDLINE | ID: mdl-37615873
The synapse is a highly specialized asymmetric structure that transmits and stores information in the brain. The size of pre- and postsynaptic structures and function is well coordinated at the individual synapse level. For example, large postsynaptic dendritic spines have a larger postsynaptic density with higher α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptor (AMPAR) number on their surface, while juxtaposing presynaptic terminals have a larger active zone and higher release probability. This indicates that pre- and postsynaptic domains bidirectionally communicate to coordinate assembly of specific molecules on both sides of the synaptic cleft. Cell adhesion molecules (CAMs) that localize at synapses form transsynaptic protein interactions across the synaptic cleft and play important roles in synapse formation and regulation. The extracellular domain of CAMs is essential for specific synapse formation and function. In contrast, the intracellular domain is necessary for binding with synaptic molecules and signal transduction. Therefore, CAMs play an essential role on synapse function and structure. In fact, ample evidence indicates that transsynaptic CAMs instruct and modulate functions at presynaptic sites. This chapter focuses on transsynaptic protein interactions that regulate presynaptic functions emphasizing the role of neuronal CAMs and the intracellular mechanism of their regulation.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Encéfalo / Moléculas de Adhesión Celular Límite: Humans Idioma: En Revista: Adv Neurobiol Año: 2023 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Encéfalo / Moléculas de Adhesión Celular Límite: Humans Idioma: En Revista: Adv Neurobiol Año: 2023 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Estados Unidos