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SEL1L-HRD1 endoplasmic reticulum-associated degradation controls STING-mediated innate immunity by limiting the size of the activable STING pool.
Ji, Yewei; Luo, Yuan; Wu, Yating; Sun, Yao; Zhao, Lianfeng; Xue, Zhen; Sun, Mengqi; Wei, Xiaoqiong; He, Zinan; Wu, Shuangcheng Alivia; Lin, Liangguang Leo; Lu, You; Chang, Lei; Chen, Fei; Chen, Siyu; Qian, Wei; Xu, Xiaoxi; Chen, Shengnuo; Pan, Dongli; Zhou, Zhangsen; Xia, Sheng; Hu, Chih-Chi Andrew; Liang, Tingbo; Qi, Ling.
Afiliación
  • Ji Y; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China. ywji@zju.edu.cn.
  • Luo Y; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI, USA. ywji@zju.edu.cn.
  • Wu Y; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China. ywji@zju.edu.cn.
  • Sun Y; Department of Hepatobiliary and Pancreatic Surgery, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Zhao L; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Xue Z; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Sun M; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Wei X; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • He Z; Graduate Program in Nutrition, Cornell University, Ithaca, NY, USA.
  • Wu SA; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Lin LL; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI, USA.
  • Lu Y; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Chang L; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI, USA.
  • Chen F; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI, USA.
  • Chen S; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI, USA.
  • Qian W; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Xu X; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Chen S; Department of Medical Microbiology and Parasitology, Zhejiang University School of Medicine, Hangzhou, China.
  • Pan D; Department of Immunology, School of Medicine, Jiangsu University, Zhenjiang, China.
  • Zhou Z; Division of Metabolism, Endocrinology and Diabetes, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, MI, USA.
  • Xia S; Zhejiang Provincial Key Laboratory of Pancreatic Disease, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.
  • Hu CA; Department of Medical Microbiology and Parasitology, Zhejiang University School of Medicine, Hangzhou, China.
  • Liang T; Department of Molecular and Integrative Physiology, University of Michigan Medical School, Ann Arbor, MI, USA.
  • Qi L; Shanghai Institute of Nutrition and Health, Chinese Academy of Sciences, Shanghai, China.
Nat Cell Biol ; 25(5): 726-739, 2023 05.
Article en En | MEDLINE | ID: mdl-37142791
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ubiquitina-Proteína Ligasas / Degradación Asociada con el Retículo Endoplásmico Tipo de estudio: Risk_factors_studies Idioma: En Revista: Nat Cell Biol Año: 2023 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ubiquitina-Proteína Ligasas / Degradación Asociada con el Retículo Endoplásmico Tipo de estudio: Risk_factors_studies Idioma: En Revista: Nat Cell Biol Año: 2023 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido