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Anticancer Activity of Novel Difluorinated Curcumin Analog and Its Inclusion Complex with 2-Hydroxypropyl-ß-Cyclodextrin against Pancreatic Cancer.
Bhattacharyya, Sangita; Ghosh, Hindole; Covarrubias-Zambrano, Obdulia; Jain, Krishan; Swamy, K Venkateswara; Kasi, Anup; Hamza, Ameer; Anant, Shrikant; VanSaun, Michael; Weir, Scott J; Bossmann, Stefan H; Padhye, Subhash B; Dandawate, Prasad.
Afiliación
  • Bhattacharyya S; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • Ghosh H; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • Covarrubias-Zambrano O; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • Jain K; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • Swamy KV; MIT School of Bioengineering, Sciences & Research, MIT Art, Design and Technology University, Pune 412201, India.
  • Kasi A; Division of Medical Oncology, University of Kansas, Kansas City, KS 66160, USA.
  • Hamza A; Pathology and Laboratory Medicine, University of Kansas, Kansas City, KS 66160, USA.
  • Anant S; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • VanSaun M; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • Weir SJ; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
  • Bossmann SH; Division of Medical Oncology, University of Kansas, Kansas City, KS 66160, USA.
  • Padhye SB; Institute for Advancing Medical Innovation, University of Kansas Medical Center, Kansas City, KS 66160, USA.
  • Dandawate P; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS 66103, USA.
Int J Mol Sci ; 24(7)2023 Mar 28.
Article en En | MEDLINE | ID: mdl-37047307
Pancreatic ductal adenocarcinoma (PDAC) is the primary reason for cancer-related deaths in the US. Genetic mutations, drug resistance, the involvement of multiple signaling pathways, cancer stem cells (CSCs), and desmoplastic stroma, which hinders drug penetrance, contribute to poor chemotherapeutic efficacy. Hence, there is a need to identify novel drugs with improved delivery to improve treatment outcomes. Curcumin is one such compound that can inhibit multiple signaling pathways and CSCs. However, curcumin's clinical applicability for treating PDAC is limited because of its poor solubility in water and metabolic instability. Hence, we developed a difluorinated curcumin (CDF) analog that accumulates selectively in the pancreas and inhibits PDAC growth in vitro and in vivo. In the present work, we developed its 2-hydroxy-propyl-ß-cyclodextrin (HCD) inclusion complex to increase its water solubility and hydrolytic stability. The CDFHCD inclusion complex was characterized by spectroscopic, thermal, and microscopic techniques. The inclusion complex exhibited increased aqueous solubility, hydrolytic stability, and antiproliferative activity compared to parent CDF. Moreover, CDF and CDFHCD inhibited colony and spheroid formation, and induced cell cycle and apoptosis in PDAC cell lines. Hence, CDFHCD self-assembly is an efficient approach to increase water solubility and anticancer therapeutic efficacy, which now warrants advancement towards a clinical proof of concept in PDAC patients.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias Pancreáticas / Curcumina / Carcinoma Ductal Pancreático Límite: Humans Idioma: En Revista: Int J Mol Sci Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias Pancreáticas / Curcumina / Carcinoma Ductal Pancreático Límite: Humans Idioma: En Revista: Int J Mol Sci Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Suiza