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Levofloxacin induces differential effects in the transcriptome between the gut, peripheral and axial joints in the Spondyloarthritis DBA/1 mice: Improvement of intestinal dysbiosis and the overall inflammatory process.
González-Chávez, Susana Aideé; Salas-Leiva, Joan S; Salas-Leiva, Dayana E; López-Loeza, Salma Marcela; Sausameda-García, Jasanai; Orrantia-Borunda, Erasmo; Burgos-Vargas, Rubén; Alvarado-Jáquez, Maria Fernanda; Torres-Quintana, Mayra; Cuevas-Martínez, Rubén; Chaparro-Barrera, Eduardo; Marín-Terrazas, Carlos; Espino-Solís, Gerardo Pável; Romero-López, José Pablo; Bernal-Alferes, Brian de Jesús; Pacheco-Tena, César.
Afiliación
  • González-Chávez SA; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Salas-Leiva JS; Departamento de Medio Ambiente y Energía, CONACyT-Centro de Investigación en Materiales Avanzados, Chihuahua, México.
  • Salas-Leiva DE; Department of Biochemistry, University of Cambridge, Cambridge, United Kingdom.
  • López-Loeza SM; Department of Biochemistry and Molecular Biology, Institute for Comparative Genomics (ICG), Dalhousie University, Halifax, NS, Canada.
  • Sausameda-García J; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Orrantia-Borunda E; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Burgos-Vargas R; Departamento de Medio Ambiente y Energía, CONACyT-Centro de Investigación en Materiales Avanzados, Chihuahua, México.
  • Alvarado-Jáquez MF; Department of Rheumatology, Hospital General de México, "Dr. Eduardo Liceaga", Ciudad de México, México.
  • Torres-Quintana M; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Cuevas-Martínez R; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Chaparro-Barrera E; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Marín-Terrazas C; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Espino-Solís GP; Facultad de Medicina y Ciencias Biomédicas, Laboratorio PABIOM, Universidad Autónoma de Chihuahua, Chihuahua, México.
  • Romero-López JP; Translational Research Laboratory and National Laboratory of Flow Cytometry, Autonomous University of Chihuahua, Circuito Universitario, Campus II, Chihuahua, Mexico.
  • Bernal-Alferes BJ; Laboratorio de Inmunología Clínica 1, Instituto Politécnico Nacional de México, Posgrado en Ciencias Quimicobiológicas, Escuela Nacional de Ciencias Biológicas, Ciudad de México, México.
  • Pacheco-Tena C; Laboratorio de Inmunología Clínica 1, Instituto Politécnico Nacional de México, Posgrado en Ciencias Quimicobiológicas, Escuela Nacional de Ciencias Biológicas, Ciudad de México, México.
PLoS One ; 18(2): e0281265, 2023.
Article en En | MEDLINE | ID: mdl-36730179
To analyze the effect of levofloxacin-induced intestinal microbiota modifications on intestinal, joint, and systemic inflammation in the DBA/1 mice with spontaneous arthritis. The study included two groups of mice, one of which received levofloxacin. The composition and structure of the microbiota were determined in the mice's stool using 16S rRNA sequencing; the differential taxa and metabolic pathway between mice treated with levofloxacin and control mice were also defied. The effect of levofloxacin was evaluated in the intestines, hind paws, and spines of mice through DNA microarray transcriptome and histopathological analyses; systemic inflammation was measured by flow cytometry. Levofloxacin decreased the pro-inflammatory bacteria, including Prevotellaceae, Odoribacter, and Blautia, and increased the anti-inflammatory Muribaculaceae in mice's stool. Histological analysis confirmed the intestinal inflammation in control mice, while in levofloxacin-treated mice, inflammation was reduced; in the hind paws and spines, levofloxacin also decreased the inflammation. Microarray showed the downregulation of genes and signaling pathways relevant in spondyloarthritis, including several cytokines and chemokines. Levofloxacin-treated mice showed differential transcriptomic profiles between peripheral and axial joints and intestines. Levofloxacin decreased the expression of TNF-α, IL-23a, and JAK3 in the three tissues, but IL-17 behaved differently in the intestine and the joints. Serum TNF-α was also reduced in levofloxacin-treated mice. Our results suggest that the microbiota modification aimed at reducing pro-inflammatory and increasing anti-inflammatory bacteria could potentially be a coadjuvant in treating inflammatory arthropathies.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Espondiloartritis / Levofloxacino Límite: Animals Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2023 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Espondiloartritis / Levofloxacino Límite: Animals Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2023 Tipo del documento: Article Pais de publicación: Estados Unidos