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Oncogenic PKA signaling increases c-MYC protein expression through multiple targetable mechanisms.
Chan, Gary K L; Maisel, Samantha; Hwang, Yeonjoo C; Pascual, Bryan C; Wolber, Rebecca R B; Vu, Phuong; Patra, Krushna C; Bouhaddou, Mehdi; Kenerson, Heidi L; Lim, Huat C; Long, Donald; Yeung, Raymond S; Sethupathy, Praveen; Swaney, Danielle L; Krogan, Nevan J; Turnham, Rigney E; Riehle, Kimberly J; Scott, John D; Bardeesy, Nabeel; Gordan, John D.
Afiliación
  • Chan GKL; Division of Hematology/Oncology, Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, San Francisco, United States.
  • Maisel S; Quantitative Biosciences Institute (QBI), University of California San Francisco, San Francisco, United States.
  • Hwang YC; Division of Hematology/Oncology, Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, San Francisco, United States.
  • Pascual BC; Quantitative Biosciences Institute (QBI), University of California San Francisco, San Francisco, United States.
  • Wolber RRB; Division of Hematology/Oncology, Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, San Francisco, United States.
  • Vu P; Quantitative Biosciences Institute (QBI), University of California San Francisco, San Francisco, United States.
  • Patra KC; Division of Hematology/Oncology, Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, San Francisco, United States.
  • Bouhaddou M; Quantitative Biosciences Institute (QBI), University of California San Francisco, San Francisco, United States.
  • Kenerson HL; Division of Hematology/Oncology, Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, San Francisco, United States.
  • Lim HC; Quantitative Biosciences Institute (QBI), University of California San Francisco, San Francisco, United States.
  • Long D; Department of Medicine, Harvard Medical School, Boston, United States.
  • Yeung RS; Massachusetts General Hospital Cancer Center, Boston, United States.
  • Sethupathy P; Department of Medicine, Harvard Medical School, Boston, United States.
  • Swaney DL; Massachusetts General Hospital Cancer Center, Boston, United States.
  • Krogan NJ; Department of Cellular and Molecular Pharmacology, University of California San Francisco, San Francisco, United States.
  • Turnham RE; J. David Gladstone Institute, San Francisco, United States.
  • Riehle KJ; Department of Surgery and Northwest Liver Research Program, University of Washington, Seattle, United States.
  • Scott JD; Division of Hematology/Oncology, Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, San Francisco, United States.
  • Bardeesy N; Quantitative Biosciences Institute (QBI), University of California San Francisco, San Francisco, United States.
  • Gordan JD; Department of Biomedical Sciences, College of Veterinary Medicine, Cornell University, New York, United States.
Elife ; 122023 01 24.
Article en En | MEDLINE | ID: mdl-36692000
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Quinasas Dependientes de AMP Cíclico / Carcinoma Hepatocelular Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Elife Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Quinasas Dependientes de AMP Cíclico / Carcinoma Hepatocelular Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Elife Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido