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MiR-182-5p Mediated by Exosomes Derived From Bone Marrow Mesenchymal Stem Cell Attenuates Inflammatory Responses by Targeting TLR4 in a Mouse Model of Myocardial Infraction.
Sun, Chuang; Li, Wei; Li, Yanhong; Chen, Jian; An, Huixian; Zeng, Guangwei; Wang, Tingting; Guo, Yazhou; Wang, Changying.
Afiliación
  • Sun C; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • Li W; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • Li Y; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • Chen J; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • An H; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • Zeng G; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • Wang T; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
  • Guo Y; College of Veterinary Medicine, Northwest A&F University, Xianyang 712100, China.
  • Wang C; Department of Cardiology, Xi'an International Medical Center Hospital, Xi'an 710100, Shaanxi, China.
Immune Netw ; 22(6): e49, 2022 Dec.
Article en En | MEDLINE | ID: mdl-36627935
Exosomes derived from mesenchymal stem cells (MSCs) could protect against myocardial infarction (MI). TLR4 is reported to play an important role in MI, while microRNA-182-5p (miR-182-5p) negatively regulates TLR4 expression. Therefore, we hypothesize that MSCs-derived exosomes overexpressing miR-182-5p may have beneficial effects on MI. We generated bone marrow mesenchymal stem cells (BM-MSCs) and overexpressed miR-182-5p in these cells for exosome isolation. H2O2-stimulated neonatal mouse ventricle myocytes (NMVMs) and MI mouse model were employed, which were subjected to exosome treatment. The expression of inflammatory factors, heart function, and TLR4 signaling pathway activation were monitored. It was found that miR-182-5p decreased TLR4 expression in BM-MSCs and NMVMs. Administration of exosomes overexpressing miR-182-5p to H2O2-stimulated NMVMs enhanced cell viability and suppressed the expression of inflammatory cytokines. In addition, they promoted heart function, suppressed inflammatory responses, and de-activated TLR4/NF-κB signaling pathway in MI mice. In conclusion, miR-182-5p transferred by the exosomes derived from BM-MSCs protected against MI-induced impairments by targeting TLR4.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Immune Netw Año: 2022 Tipo del documento: Article País de afiliación: China Pais de publicación: Corea del Sur

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Immune Netw Año: 2022 Tipo del documento: Article País de afiliación: China Pais de publicación: Corea del Sur