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Vascular injury is associated with repetitive head impacts and tau pathology in chronic traumatic encephalopathy.
Kirsch, Daniel; Shah, Arsal; Dixon, Erin; Kelley, Hunter; Cherry, Jonathan D; Xia, Weiming; Daley, Sarah; Aytan, Nurgul; Cormier, Kerry; Kubilus, Carol; Mathias, Rebecca; Alvarez, Victor E; Huber, Bertrand R; McKee, Ann C; Stein, Thor D.
Afiliación
  • Kirsch D; Boston University Alzheimer's Disease and CTE Center, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Shah A; VA Boston Healthcare System, Boston, Massachusetts, USA.
  • Dixon E; Department of Pathology and Laboratory Medicine, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Kelley H; Boston University Alzheimer's Disease and CTE Center, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Cherry JD; VA Boston Healthcare System, Boston, Massachusetts, USA.
  • Xia W; VA Bedford Healthcare System, Bedford, Massachusetts, USA.
  • Daley S; Boston University Alzheimer's Disease and CTE Center, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Aytan N; VA Boston Healthcare System, Boston, Massachusetts, USA.
  • Cormier K; VA Bedford Healthcare System, Bedford, Massachusetts, USA.
  • Kubilus C; Boston University Alzheimer's Disease and CTE Center, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Mathias R; VA Boston Healthcare System, Boston, Massachusetts, USA.
  • Alvarez VE; Boston University Alzheimer's Disease and CTE Center, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Huber BR; VA Boston Healthcare System, Boston, Massachusetts, USA.
  • McKee AC; Department of Pathology and Laboratory Medicine, Boston University School of Medicine, Boston, Massachusetts, USA.
  • Stein TD; Boston University Alzheimer's Disease and CTE Center, Boston University School of Medicine, Boston, Massachusetts, USA.
J Neuropathol Exp Neurol ; 82(2): 127-139, 2023 01 20.
Article en En | MEDLINE | ID: mdl-36617181
Chronic traumatic encephalopathy (CTE) is a neurodegenerative disease linked to repetitive head impacts (RHI) and characterized by perivascular hyperphosphorylated tau (p-tau) deposits. The role of vascular injury, blood-brain barrier leakage, and neuroinflammation in CTE pathogenesis is not well understood. We performed quantitative immunoassays for intercellular adhesion molecule 1 (ICAM1), vascular cellular adhesion molecule 1 (VCAM1), and C-reactive protein (CRP) within the postmortem dorsolateral frontal cortex of participants with and without a history of RHI and CTE (n = 156), and tested for associations with RHI, microgliosis, and tau pathology measures. Levels of vascular injury-associated markers ICAM1, VCAM1, and CRP were increased in CTE compared to RHI-exposed and -naïve controls. ICAM1 and CRP increased with RHI exposure duration (p < 0.01) and were associated with increased microglial density (p < 0.001) and tau pathology (AT8, p-tau396, p-tau202; p < 0.05). Histologically, there was significantly increased ICAM1 staining of the microvasculature, extracellular space, and astrocytes at the sulcal depths in high stage CTE compared to both low stage CTE and controls. Multifocal perivascular immunoreactivity for serum albumin was present in all RHI-exposed individuals. These findings demonstrate that vascular injury markers are associated with RHI exposure, duration, and microgliosis, are elevated in CTE, and increase with disease severity.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedades Neurodegenerativas / Lesiones del Sistema Vascular / Encefalopatía Traumática Crónica Tipo de estudio: Risk_factors_studies Límite: Humans Idioma: En Revista: J Neuropathol Exp Neurol Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedades Neurodegenerativas / Lesiones del Sistema Vascular / Encefalopatía Traumática Crónica Tipo de estudio: Risk_factors_studies Límite: Humans Idioma: En Revista: J Neuropathol Exp Neurol Año: 2023 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido