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AJUBA and WTIP can compete with LIMD1 for junctional localization and LATS regulation.
Kirichenko, Elmira; Irvine, Kenneth D.
Afiliación
  • Kirichenko E; Waksman Institute and Department of Molecular Biology and Biochemistry, Rutgers University, 190 Frelinghusen Rd, Piscataway NJ 08854 USA.
  • Irvine KD; Waksman Institute and Department of Molecular Biology and Biochemistry, Rutgers University, 190 Frelinghusen Rd, Piscataway NJ 08854 USA.
MicroPubl Biol ; 20222022.
Article en En | MEDLINE | ID: mdl-36439396
Each of the three mammalian Ajuba family proteins, AJUBA, LIMD1 and WTIP, exhibit tension-dependent localization to adherens junctions, and can associate with Lats kinases. However, only LIMD1 has been directly demonstrated to directly regulate Lats activity in vivo. To assess the relationship of LIMD1 to AJUBA and WTIP, and the potential contributions of AJUBA and WTIP to Lats regulation, we examined the consequences of over-expressing AJUBA and WTIP in MCF10A cells. Over-expression of either AJUBA or WTIP reduced junctional localization of LIMD1, implying that these proteins can compete for binding to adherens junctions. This over-expression also reduced junctional localization of LATS1, implying that AJUBA or WTIP are unable to efficiently recruit Lats kinases to adherens junctions. This over-expression was also associated with increased YAP1 phosphorylation and decreased YAP1 nuclear localization, consistent with increased Lats kinase activity. These observations indicate that AJUBA and WTIP compete with LIMD1 for association with adherens junctions but have activities distinct from LIMD1 in Hippo pathway regulation. They further suggest that the ability of Ajuba family proteins to associate with Lats kinases in solution is not sufficient to enable regulation in vivo, and that tumor suppressor activities of AJUBA and WTIP could stem in part from competition with LIMD1 for regulation of Lats kinases at cell junctions.

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: MicroPubl Biol Año: 2022 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: MicroPubl Biol Año: 2022 Tipo del documento: Article Pais de publicación: Estados Unidos