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Phenotypic and Genotypic Characterization of Hereditary Angioedema in Saudi Arabia.
Sheikh, Farrukh; Alajlan, Huda; Albanyan, Maram; Alruwaili, Hibah; Alawami, Fatimah; Sumayli, Safia; Al Gazlan, Sulaiman; Abu Awwad, Sawsan; Al-Dhekri, Hasan; Al-Saud, Bandar; Arnaout, Rand; Alrayes, Hassan; Sayes, Najla; Al-Hamed, Mohamed H; Al-Mousa, Hamoud; AlShareef, Saad; Alazami, Anas M.
Afiliación
  • Sheikh F; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Alajlan H; Translational Genomics Department, Centre for Genomic Medicine, King Faisal Specialist Hospital & Research Centre, MBC 3, P.O. Box 3354, Riyadh, 11211, Saudi Arabia.
  • Albanyan M; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Alruwaili H; Translational Genomics Department, Centre for Genomic Medicine, King Faisal Specialist Hospital & Research Centre, MBC 3, P.O. Box 3354, Riyadh, 11211, Saudi Arabia.
  • Alawami F; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Sumayli S; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Al Gazlan S; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Abu Awwad S; Department of Pediatrics, Allergy and Immunology Section, Faisal Specialist Hospital and Research Centre, Riyadh, King, Saudi Arabia.
  • Al-Dhekri H; Department of Pediatrics, Allergy and Immunology Section, Faisal Specialist Hospital and Research Centre, Riyadh, King, Saudi Arabia.
  • Al-Saud B; Department of Pediatrics, Allergy and Immunology Section, Faisal Specialist Hospital and Research Centre, Riyadh, King, Saudi Arabia.
  • Arnaout R; College of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
  • Alrayes H; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Sayes N; Department of Medicine, Allergy and Immunology Section, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • Al-Hamed MH; Department of Medicine, King Faisal Specialist Hospital and Research Centre, Jeddah, Saudi Arabia.
  • Al-Mousa H; Clinical Genomics Department, Centre for Genomic Medicine, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
  • AlShareef S; Department of Pediatrics, Allergy and Immunology Section, Faisal Specialist Hospital and Research Centre, Riyadh, King, Saudi Arabia.
  • Alazami AM; College of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
J Clin Immunol ; 43(2): 479-484, 2023 02.
Article en En | MEDLINE | ID: mdl-36348183
Hereditary angioedema (HAE) is a potentially life-threatening autosomal dominant disorder affecting roughly 1:50,000 individuals. It is commonly characterized by swelling of the larynx, gastrointestinal tract, extremities, and skin. There is growing genetic heterogeneity associated with this disease but more than 95% of mutations are found in SERPING1, the gene which encodes complement 1 inhibitor (C1-INH). HAE cohorts from several populations have been published but no large scale study has been reported from the Arab world to date. Here we document the clinical and genetic findings of HAE patients from a single Saudi institution, which is a major referral center at the national level. A total of 51 patients across 17 unrelated families were recruited including two large multi-generational families, of which one contained an in-frame exonic deletion that was resolved through MLPA. Two cases were negative for all the genes we tested (including F12, PLG, ANGPT1, MYOF, KNG1, and HS3ST6). The predominant HAE subtype in our cohort was type I, at 76%. We were able to uncover a mutation in 49 patients (96%). No type III (normal C1-INH) patients were encountered in the clinic, suggesting that this subtype does not play a major role in HAE pathogenesis in Saudi Arabia. Additionally, the existence of four patients with consistently normal complement 4 (C4) levels alongside abnormal C1-INH profiles highlights the utility of dual screening for both proteins in suspected patients.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Angioedemas Hereditarios Límite: Humans País/Región como asunto: Asia Idioma: En Revista: J Clin Immunol Año: 2023 Tipo del documento: Article País de afiliación: Arabia Saudita Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Angioedemas Hereditarios Límite: Humans País/Región como asunto: Asia Idioma: En Revista: J Clin Immunol Año: 2023 Tipo del documento: Article País de afiliación: Arabia Saudita Pais de publicación: Países Bajos