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Inverted direct allorecognition triggers early donor-specific antibody responses after transplantation.
Charmetant, Xavier; Chen, Chien-Chia; Hamada, Sarah; Goncalves, David; Saison, Carole; Rabeyrin, Maud; Rabant, Marion; Duong van Huyen, Jean-Paul; Koenig, Alice; Mathias, Virginie; Barba, Thomas; Lacaille, Florence; le Pavec, Jérôme; Brugière, Olivier; Taupin, Jean-Luc; Chalabreysse, Lara; Mornex, Jean-François; Couzi, Lionel; Graff-Dubois, Stéphanie; Jeger-Madiot, Raphaël; Tran-Dinh, Alexy; Mordant, Pierre; Paidassi, Helena; Defrance, Thierry; Morelon, Emmanuel; Badet, Lionel; Nicoletti, Antonino; Dubois, Valérie; Thaunat, Olivier.
Afiliación
  • Charmetant X; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
  • Chen CC; Department of Surgery, National Taiwan University Hospital, Taipei 100, Taiwan.
  • Hamada S; French National Blood Service (EFS), HLA Laboratory, 69150 Décines, France.
  • Goncalves D; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
  • Saison C; French National Blood Service (EFS), HLA Laboratory, 69150 Décines, France.
  • Rabeyrin M; Department of Pathology, Hospices Civils de Lyon, Groupement Hospitalier Est, 69500 Bron, France.
  • Rabant M; Pathology Department, Assistance Publique-Hôpitaux de Paris, Hôpital Necker, 75015 Paris, France.
  • Duong van Huyen JP; Pathology Department, Assistance Publique-Hôpitaux de Paris, Hôpital Necker, 75015 Paris, France.
  • Koenig A; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
  • Mathias V; Lyon-Est Medical Faculty, Claude Bernard University (Lyon 1), 69008 Lyon, France.
  • Barba T; Department of Transplantation, Nephrology and Clinical Immunology, Hospices Civils de Lyon, Edouard Herriot Hospital, 69003 Lyon, France.
  • Lacaille F; French National Blood Service (EFS), HLA Laboratory, 69150 Décines, France.
  • le Pavec J; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
  • Brugière O; Pediatric Gastroenterology-Hepatology-Nutrition Unit, Hôpital Universitaire Necker-Enfants malades, 75015 Paris, France.
  • Taupin JL; Department of Pulmonology and Lung Transplantation, Marie Lannelongue Hospital, 92350 Le Plessis Robinson, France.
  • Chalabreysse L; Pulmonology Department, Adult Cystic Fibrosis Centre and Lung Transplantation Department, Foch Hospital, 92150 Suresnes, France.
  • Mornex JF; Laboratory of Immunology and Histocompatibility, Hôpital Saint-Louis APHP, 75010 Paris, France.
  • Couzi L; INSERM U976 Institut de Recherche Saint-Louis, Université Paris Diderot, 75010 Paris, France.
  • Graff-Dubois S; Department of Pathology, Hospices Civils de Lyon, Groupement Hospitalier Est, 69500 Bron, France.
  • Jeger-Madiot R; Université de Lyon, Université Lyon 1, INRAE, IVPC, UMR754, 69000 Lyon, France.
  • Tran-Dinh A; Department of Pneumology, GHE, Hospices Civils de Lyon, 69000 Lyon, France.
  • Mordant P; Department of Nephrology, Transplantation, Dialysis, Apheresis, Pellegrin Hospital, 33000 Bordeaux, France.
  • Paidassi H; Sorbonne Université, INSERM, Immunology-Immunopathology-Immunotherapy (i3), 75013 Paris, France.
  • Defrance T; Sorbonne Université, INSERM, Immunology-Immunopathology-Immunotherapy (i3), 75013 Paris, France.
  • Morelon E; Université de Paris, LVTS, INSERM U1148, 75018 Paris, France.
  • Badet L; Department of Vascular and Thoracic Surgery, Assistance Publique-Hôpitaux de Paris, Bichat-Claude Bernard Hospital, 75018 Paris, France.
  • Nicoletti A; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
  • Dubois V; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
  • Thaunat O; CIRI, Centre International de Recherche en Infectiologie, Université de Lyon, INSERM U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, ENS de Lyon, 69007 Lyon, France.
Sci Transl Med ; 14(663): eabg1046, 2022 Sep 21.
Article en En | MEDLINE | ID: mdl-36130013
The generation of antibodies against donor-specific major histocompatibility complex (MHC) antigens, a type of donor-specific antibodies (DSAs), after transplantation requires that recipient's allospecific B cells receive help from T cells. The current dogma holds that this help is exclusively provided by the recipient's CD4+ T cells that recognize complexes of recipient's MHC II molecules and peptides derived from donor-specific MHC alloantigens, a process called indirect allorecognition. Here, we demonstrated that, after allogeneic heart transplantation, CD3ε knockout recipient mice lacking T cells generate a rapid, transient wave of switched alloantibodies, predominantly directed against MHC I molecules. This is due to the presence of donor CD4+ T cells within the graft that recognize intact recipient's MHC II molecules expressed by B cell receptor-activated allospecific B cells. Indirect evidence suggests that this inverted direct pathway is also operant in patients after transplantation. Resident memory donor CD4+ T cells were observed in perfusion liquids of human renal and lung grafts and acquired B cell helper functions upon in vitro stimulation. Furthermore, T follicular helper cells, specialized in helping B cells, were abundant in mucosa-associated lymphoid tissue of lung and intestinal grafts. In the latter, more graft-derived passenger T cells correlated with the detection of donor T cells in recipient's circulation; this, in turn, was associated with an early transient anti-MHC I DSA response and worse transplantation outcomes. We conclude that this inverted direct allorecognition is a possible explanation for the early transient anti-MHC DSA responses frequently observed after lung or intestinal transplantations.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Isoanticuerpos / Formación de Anticuerpos Límite: Animals / Humans Idioma: En Revista: Sci Transl Med Asunto de la revista: CIENCIA / MEDICINA Año: 2022 Tipo del documento: Article País de afiliación: Francia Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Isoanticuerpos / Formación de Anticuerpos Límite: Animals / Humans Idioma: En Revista: Sci Transl Med Asunto de la revista: CIENCIA / MEDICINA Año: 2022 Tipo del documento: Article País de afiliación: Francia Pais de publicación: Estados Unidos