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Preventive Effects of l-Glutamine on High-Fat Diet-Induced Metabolic Disorders Linking with Regulation of Intestinal Barrier Integrity, Hepatic Lipid Metabolism, and Gut Microbiota in Rats.
He, Yu; Song, Zhuan; Ji, Yun; Tso, Patrick; Wu, Zhenlong.
Afiliación
  • He Y; State Key Laboratory of Animal Nutrition, China Agricultural University, Beijing 100193, China.
  • Song Z; State Key Laboratory of Animal Nutrition, China Agricultural University, Beijing 100193, China.
  • Ji Y; State Key Laboratory of Animal Nutrition, China Agricultural University, Beijing 100193, China.
  • Tso P; Department of Pathology and Laboratory Medicine, Metabolic Diseases Institute, University of Cincinnati, Cincinnati, 2120 E. Galbraith Road, Building A, Cincinnati, Ohio 45237, United States.
  • Wu Z; State Key Laboratory of Animal Nutrition, China Agricultural University, Beijing 100193, China.
J Agric Food Chem ; 70(38): 11923-11934, 2022 Sep 28.
Article en En | MEDLINE | ID: mdl-36122193
The present study was conducted to investigate the effects of l-glutamine (Gln) on a high-fat diet (HFD)-induced lipid metabolic abnormality and explore its possible mechanisms. The results demonstrated that Gln administration reduced body weight, improved serum lipids, and decreased glucose tolerance in HFD-fed rats. Meanwhile, Gln administration alleviated liver injury, reduced the hepatic inflammatory response by inhibiting NLRP3 inflammasome activation, and decreased hepatic lipid accumulation by promoting VLDL secretion and fatty acid ß-oxidation, as well as reduced bile acid synthesis by activating hepatic and ileal FXR in HFD-fed rats. Moreover, Gln administration restored HFD-induced intestinal barrier dysfunction, promoted intestinal fat absorption, suppressed intestinal inflammation, and also reshaped the gut microbiota composition in HFD-fed rats by downregulating the abundance of potential pathogens Escherichia-Shigella and upregulating the abundance of beneficial bacteria such as Akkermansia. To conclude, the present results showed that Gln may be a potential option for preventing HFD-induced metabolic disorders via the gut-liver axis.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Microbioma Gastrointestinal / Enfermedades Intestinales / Enfermedades Metabólicas Límite: Animals Idioma: En Revista: J Agric Food Chem Año: 2022 Tipo del documento: Article País de afiliación: China Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Microbioma Gastrointestinal / Enfermedades Intestinales / Enfermedades Metabólicas Límite: Animals Idioma: En Revista: J Agric Food Chem Año: 2022 Tipo del documento: Article País de afiliación: China Pais de publicación: Estados Unidos