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Structure of full-length TSH receptor in complex with antibody K1-70™.
Núñez Miguel, Ricardo; Sanders, Paul; Allen, Lloyd; Evans, Michele; Holly, Matthew; Johnson, William; Sullivan, Andrew; Sanders, Jane; Furmaniak, Jadwiga; Rees Smith, Bernard.
Afiliación
  • Núñez Miguel R; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Sanders P; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Allen L; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Evans M; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Holly M; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Johnson W; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Sullivan A; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Sanders J; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Furmaniak J; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
  • Rees Smith B; FIRS Laboratories, RSR Ltd, Parc Ty Glas, Llanishen, Cardiff, UK.
J Mol Endocrinol ; 70(1)2023 01 01.
Article en En | MEDLINE | ID: mdl-36069797
Determination of the full-length thyroid-stimulating hormone receptor (TSHR) structure by cryo-electron microscopy (cryo-EM) is described. The TSHR complexed with human monoclonal TSHR autoantibody K1-70™ (a powerful inhibitor of TSH action) was detergent solubilised, purified to homogeneity and analysed by cryo-EM. The structure (global resolution 3.3 Å) is a monomer with all three domains visible: leucine-rich domain (LRD), hinge region (HR) and transmembrane domain (TMD). The TSHR extracellular domain (ECD, composed of the LRD and HR) is positioned on top of the TMD extracellular surface. Extensive interactions between the TMD and ECD are observed in the structure, and their analysis provides an explanation of the effects of various TSHR mutations on TSHR constitutive activity and on ligand-induced activation. K1-70™ is seen to be well clear of the lipid bilayer. However, superimposition of M22™ (a human monoclonal TSHR autoantibody which is a powerful stimulator of the TSHR) on the cryo-EM structure shows that it would clash with the bilayer unless the TSHR HR rotates upwards as part of the M22™ binding process. This rotation could have an important role in TSHR stimulation by M22™ and as such provides an explanation as to why K1-70™ blocks the binding of TSH and M22™ without activating the receptor itself.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Autoanticuerpos / Receptores de Tirotropina Límite: Humans Idioma: En Revista: J Mol Endocrinol Asunto de la revista: BIOLOGIA MOLECULAR / ENDOCRINOLOGIA Año: 2023 Tipo del documento: Article Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Autoanticuerpos / Receptores de Tirotropina Límite: Humans Idioma: En Revista: J Mol Endocrinol Asunto de la revista: BIOLOGIA MOLECULAR / ENDOCRINOLOGIA Año: 2023 Tipo del documento: Article Pais de publicación: Reino Unido