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Energy metabolism as a target for cyclobenzaprine: A drug candidate against Visceral Leishmaniasis.
Lima, Marta Lopes; Abengózar, Maria A; Torres-Santos, Eduardo Caio; Borborema, Samanta Etel Treiger; Godzien, Joanna; López-Gonzálvez, Ángeles; Barbas, Coral; Rivas, Luis; Tempone, Andre Gustavo.
Afiliación
  • Lima ML; Centre for Parasitology and Mycology, Instituto Adolfo Lutz, São Paulo, São Paulo, Brazil.
  • Abengózar MA; Centro de Investigaciones Biológicas Margarita Salas (CSIC), Madrid, Spain.
  • Torres-Santos EC; Laboratório de Bioquímica de Tripanosomatídeos, Instituto Oswaldo Cruz, Fiocruz, Rio de Janeiro, Brazil.
  • Borborema SET; Centre for Parasitology and Mycology, Instituto Adolfo Lutz, São Paulo, São Paulo, Brazil.
  • Godzien J; Centre for Metabolomics and Bioanalysis (CEMBIO), Faculty of Pharmacy, Universidad CEU San Pablo, Madrid, Spain.
  • López-Gonzálvez Á; Centre for Metabolomics and Bioanalysis (CEMBIO), Faculty of Pharmacy, Universidad CEU San Pablo, Madrid, Spain.
  • Barbas C; Centre for Metabolomics and Bioanalysis (CEMBIO), Faculty of Pharmacy, Universidad CEU San Pablo, Madrid, Spain. Electronic address: cbarbas@ceu.es.
  • Rivas L; Centro de Investigaciones Biológicas Margarita Salas (CSIC), Madrid, Spain. Electronic address: luis.rivas@cib.csic.es.
  • Tempone AG; Centre for Parasitology and Mycology, Instituto Adolfo Lutz, São Paulo, São Paulo, Brazil. Electronic address: andre.tempone@ial.sp.gov.br.
Bioorg Chem ; 127: 106009, 2022 10.
Article en En | MEDLINE | ID: mdl-35841672
Leishmaniases have a broad spectrum of clinical manifestations, ranging from a cutaneous to a progressive and fatal visceral disease. Chemotherapy is nowadays the almost exclusive way to fight the disease but limited by its scarce therapeutic arsenal, on its own compromised by adverse side effects and clinical resistance. Cyclobenzaprine (CBP), an FDA-approved oral muscle relaxant drug has previously demonstrated in vitro and in vivo activity against Leishmania sp., but its targets were not fully unveiled. This study aimed to define the role of energy metabolism as a target for the leishmanicidal mechanisms of CBP. Methodology to assess CBP leishmanicidal mechanism variation of intracellular ATP levels using living Leishmania transfected with a cytoplasmic luciferase. Induction of plasma membrane permeability by assessing depolarization with DiSBAC(2)3 and entrance of the vital dye SYTOX® Green. Mitochondrial depolarization by rhodamine 123 accumulation. Mapping target site within the respiratory chain by oxygen consumption rate. Reactive oxygen species (ROS) production using MitoSOX. Morphological changes by transmission electron microscopy. CBP caused on L. infantum promastigotes a decrease of intracellular ATP levels, with irreversible depolarization of plasma membrane, the collapse of the mitochondrial electrochemical potential, mild uncoupling of the respiratory chain, and ROS production, with ensuing intracellular Ca2+ imbalance and DNA fragmentation. Electron microscopy supported autophagic features but not a massive plasma membrane disruption. The severe and irreversible mitochondrial damage induced by CBP endorsed the bioenergetics metabolism as a relevant target within the lethal programme induced by CBP in Leishmania. This, together with the mild-side effects of this oral drug, endorses CBP as an appealing novel candidate as a leishmanicidal drug under a drug repurposing strategy.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Leishmania infantum / Leishmaniasis Visceral / Antiprotozoarios Límite: Animals / Humans Idioma: En Revista: Bioorg Chem Año: 2022 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Leishmania infantum / Leishmaniasis Visceral / Antiprotozoarios Límite: Animals / Humans Idioma: En Revista: Bioorg Chem Año: 2022 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Estados Unidos