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In silico exploration of amyloid-related imaging abnormalities in the gantenerumab open-label extension trials using a semi-mechanistic model.
Aldea, Roxana; Grimm, Hans Peter; Gieschke, Ronald; Hofmann, Carsten; Lott, Dominik; Bullain, Szofia; Delmar, Paul; Klein, Gregory; Lyons, Marco; Piazza, Fabrizio; Carare, Roxana O; Mazer, Norman A.
Afiliación
  • Aldea R; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
  • Grimm HP; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
  • Gieschke R; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
  • Hofmann C; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
  • Lott D; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
  • Bullain S; Roche Product Development Neuroscience Basel Switzerland.
  • Delmar P; Roche Product Development Neuroscience Basel Switzerland.
  • Klein G; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
  • Lyons M; Roche Products Ltd Welwyn Garden City UK.
  • Piazza F; School of Medicine Laboratory of CAA and AD Translational Research and Biomarkers University of Milano-Bicocca Monza Italy.
  • Carare RO; Faculty of Medicine Interdisciplinary Dementia and Aging Centre University of Southampton Southampton UK.
  • Mazer NA; Roche Pharma Research and Early Development Roche Innovation Center Basel Switzerland.
Alzheimers Dement (N Y) ; 8(1): e12306, 2022.
Article en En | MEDLINE | ID: mdl-35676943
Introduction: Amyloid-related imaging abnormalities with edema/effusion (ARIA-E) are commonly observed with anti-amyloid therapies in Alzheimer's disease. We developed a semi-mechanistic, in silico model to understand the time course of ARIA-E and its dose dependency. Methods: Dynamic and statistical analyses of data from 112 individuals that experienced ARIA-E in the open-label extension of SCarlet RoAD (a study of gantenerumab in participants with prodromal Alzheimer's disease) and Marguerite RoAD (as study of Gantenerumab in participants with mild Alzheimer's disease) studies were used for model building. Gantenerumab pharmacokinetics, local amyloid removal, disturbance and repair of the vascular wall, and ARIA-E magnitude were represented in the novel vascular wall disturbance (VWD) model of ARIA-E. Results: The modeled individual-level profiles provided a good representation of the observed pharmacokinetics and time course of ARIA-E magnitude. ARIA-E dynamics were shown to depend on the interplay between drug-mediated amyloid removal and intrinsic vascular repair processes. Discussion: Upon further refinement and validation, the VWD model could inform strategies for dosing and ARIA monitoring in individuals with an ARIA-E history.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Alzheimers Dement (N Y) Año: 2022 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Prognostic_studies Idioma: En Revista: Alzheimers Dement (N Y) Año: 2022 Tipo del documento: Article Pais de publicación: Estados Unidos