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Importance of crosstalk between the microbiota and the neuroimmune system for tissue homeostasis.
Li, Kunyu; Ly, Kevin; Mehta, Sunali; Braithwaite, Antony.
Afiliación
  • Li K; Department of Pathology Dunedin School of Medicine University of Otago Dunedin New Zealand.
  • Ly K; Department of Pathology Dunedin School of Medicine University of Otago Dunedin New Zealand.
  • Mehta S; Department of Pathology Dunedin School of Medicine University of Otago Dunedin New Zealand.
  • Braithwaite A; Department of Pathology Dunedin School of Medicine University of Otago Dunedin New Zealand.
Clin Transl Immunology ; 11(5): e1394, 2022.
Article en En | MEDLINE | ID: mdl-35620584
The principal function of inflammation is cellular defence against 'danger signals' such as tissue injury and pathogen infection to maintain the homeostasis of the organism. The initiation and progression of inflammation are not autonomous as there is substantial evidence that inflammation is known to be strongly influenced by 'neuroimmune crosstalk', involving the production and expression of soluble signalling molecules that interact with cell surface receptors. In addition, microbiota have been found to be involved in the development and function of the nervous and immune systems and play an important role in health and disease. Herein, we provide an outline of the mechanisms of neuroimmune communication in the regulation of inflammation and immune response and then provide evidence for the involvement of microbiota in the development and functions of the host nervous and immune systems. It appears that the nervous and immune systems in multicellular organisms have co-evolved with the microbiota, such that all components are in communication to maximise the ability of the organism to adapt to a wide range of environmental stresses to maintain or restore tissue homeostasis.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Clin Transl Immunology Año: 2022 Tipo del documento: Article Pais de publicación: Australia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Clin Transl Immunology Año: 2022 Tipo del documento: Article Pais de publicación: Australia