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The Origins, Evolution, and Future of Dietary Methionine Restriction.
Fang, Han; Stone, Kirsten P; Wanders, Desiree; Forney, Laura A; Gettys, Thomas W.
Afiliación
  • Fang H; Laboratory of Nutrient Sensing and Adipocyte Signaling, Pennington Biomedical Research Center, Baton Rouge, Louisiana, USA; email: Thomas.Gettys@pbrc.edu.
  • Stone KP; Laboratory of Nutrient Sensing and Adipocyte Signaling, Pennington Biomedical Research Center, Baton Rouge, Louisiana, USA; email: Thomas.Gettys@pbrc.edu.
  • Wanders D; Department of Nutrition, Georgia State University, Atlanta, Georgia, USA.
  • Forney LA; Department of Kinesiology, Houston Baptist University, Houston, Texas, USA.
  • Gettys TW; Laboratory of Nutrient Sensing and Adipocyte Signaling, Pennington Biomedical Research Center, Baton Rouge, Louisiana, USA; email: Thomas.Gettys@pbrc.edu.
Annu Rev Nutr ; 42: 201-226, 2022 08 22.
Article en En | MEDLINE | ID: mdl-35588443
The original description of dietary methionine restriction (MR) used semipurified diets to limit methionine intake to 20% of normal levels, and this reduction in dietary methionine increased longevity by ∼30% in rats. The MR diet also produces paradoxical increases in energy intake and expenditure and limits fat deposition while reducing tissue and circulating lipids and enhancing overall insulin sensitivity. In the years following the original 1993 report, a comprehensive effort has been made to understand the nutrient sensing and signaling systems linking reduced dietary methionine to the behavioral, physiological, biochemical, and transcriptional components of the response. Recent work has shown that transcriptional activation of hepatic fibroblast growth factor 21 (FGF21) is a key event linking the MR diet to many but not all components of its metabolic phenotype. These findings raise the interesting possibility of developing therapeutic, MR-based diets that produce the beneficial effects of FGF21 by nutritionally modulating its transcription and release.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Resistencia a la Insulina / Metionina Límite: Animals / Humans Idioma: En Revista: Annu Rev Nutr Año: 2022 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Resistencia a la Insulina / Metionina Límite: Animals / Humans Idioma: En Revista: Annu Rev Nutr Año: 2022 Tipo del documento: Article Pais de publicación: Estados Unidos