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The biallelic novel pathogenic variants in AGL gene in a chinese patient with glycogen storage disease type III.
Wang, Jing; Yu, Yuping; Cai, Chunquan; Zhi, Xiufang; Zhang, Ying; Zhao, Yu; Shu, Jianbo.
Afiliación
  • Wang J; Department of Gastroenterology, Tianjin Children's Hospital, 300134, Tianjin, China.
  • Yu Y; Tianjin Children's Hospital (Children's Hospital of Tianjin University), 300134, Tianjin, China.
  • Cai C; Tianjin Children's Hospital (Children's Hospital of Tianjin University), 300134, Tianjin, China.
  • Zhi X; Graduate College of Tianjin Medical University, 300070, Tianjin, China.
  • Zhang Y; Tianjin Children's Hospital (Children's Hospital of Tianjin University), 300134, Tianjin, China.
  • Zhao Y; Tianjin Pediatric Research Institute, 300134, Tianjin, China.
  • Shu J; Tianjin Key Laboratory of Birth Defects for Prevention and Treatment, 300134, Tianjin, China.
BMC Pediatr ; 22(1): 284, 2022 05 16.
Article en En | MEDLINE | ID: mdl-35578201
BACKGROUND: Glycogen storage disease type III (GSD III) is a rare autosomal recessive glycogenolysis disorder due to AGL gene variants, characterized by hepatomegaly, fasting hypoglycemia, hyperlipidemia, elevated hepatic transaminases, growth retardation, progressive myopathy, and cardiomyopathy. However, it is not easy to make a definite diagnosis in early stage of disease only based on the clinical phenotype and imageology due to its clinical heterogeneity. CASE PRESENTATION: We report a two-year-old girl with GSD III from a nonconsanguineous Chinese family, who presented with hepatomegaly, fasting hypoglycemia, hyperlipidemia, elevated levels of transaminases. Accordingly, Sanger sequencing, whole­exome sequencing of family trios, and qRT-PCR was performed, which revealed that the patient carried the compound heterogeneous variants, a novel frameshift mutation c.597delG (p. Q199Hfs*2) and a novel large gene fragment deletion of the entire exon 13 in AGL gene. The deletion of AGL was inherited from the proband's father and the c.597delG variant was from the mother. CONCLUSIONS: In this study, we identified two novel variants c.597delG (p. Q199Hfs*2) and deletion of the entire exon 13 in AGL in a Chinese GSD III patient. We extend the mutation spectrum of AGL. We suggest that high-throughput sequencing technology can detect and screen pathogenic variant, which is a scientific basis about genetic counseling and clinical diagnosis.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad del Almacenamiento de Glucógeno Tipo III / Hipoglucemia Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Humans País/Región como asunto: Asia Idioma: En Revista: BMC Pediatr Asunto de la revista: PEDIATRIA Año: 2022 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad del Almacenamiento de Glucógeno Tipo III / Hipoglucemia Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Humans País/Región como asunto: Asia Idioma: En Revista: BMC Pediatr Asunto de la revista: PEDIATRIA Año: 2022 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido