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Nesfatin-1 treatment preserves antioxidant status and attenuates renal fibrosis in rats with unilateral ureteral obstruction.
Tezcan, Neslihan; Özdemir-Kumral, Zarife Nigâr; Özkan Yenal, Naziye; Çilingir-Kaya, Özlem T; Virlan, Aysin Tulunay; Özbeyli, Dilek; Çetinel, Sule; Yegen, Berrak Ç; Koç, Mehmet.
Afiliación
  • Tezcan N; Department of Internal Medicine, Marmara University School of Medicine, Istanbul, Turkey.
  • Özdemir-Kumral ZN; Department of Physiology, Marmara University School of Medicine, Istanbul, Turkey.
  • Özkan Yenal N; Department of Pathology Laboratory Techniques, Marmara University Vocational School of Health Services, Istanbul, Turkey.
  • Çilingir-Kaya ÖT; Department of Histology & Embryology, Marmara University School of Medicine, Istanbul, Turkey.
  • Virlan AT; Department of Immunology, Marmara University School of Medicine, Istanbul, Turkey.
  • Özbeyli D; Department of Pathology Laboratory Techniques, Marmara University Vocational School of Health Services, Istanbul, Turkey.
  • Çetinel S; Department of Histology & Embryology, Marmara University School of Medicine, Istanbul, Turkey.
  • Yegen BÇ; Department of Physiology, Marmara University School of Medicine, Istanbul, Turkey.
  • Koç M; Department of Physiology, Marmara University School of Medicine, Istanbul, Turkey.
Nephrol Dial Transplant ; 37(7): 1238-1248, 2022 06 23.
Article en En | MEDLINE | ID: mdl-35218196
BACKGROUND: Nesfatin-1 (NES-1), an anorexigenic peptide, was reported to have anti-inflammatory and anti-apoptotic actions in several inflammation models. METHODS: To elucidate potential renoprotective effects of NES-1, unilateral ureteral obstruction (UUO) was induced in male Sprague Dawley rats by ligating left ureters. The rats were injected intraperitoneally with either saline (SL) or NES-1 (10 µg/kg/day) for 7 or 14 days (n = 8 in each group). On the 7th or 14th day, obstructed kidneys were removed for the isolation of leucocytes for flow-cytometric analysis and the assessments of biochemical and histopathological changes. RESULTS: Opposite to glutathione levels, renal myeloperoxidase activity in the SL-treated UUO group was significantly increased compared with the sham-operated group, while NES-1 treatment abolished the elevation. The percentages of CD8+/CD4+ T-lymphocytes infiltrating the obstructed kidneys were increased in the SL-treated groups but treatment with NES-1 did not prevent lymphocyte infiltration. Elevated tumour necrosis factor-alpha (TNF-α) levels in SL-treated UUO group were decreased with NES-1. Although total degeneration scores were similarly increased in all UUO groups, tubular dilatation scores were significantly increased in UUO groups and lowered by NES-1 only in the 7-day treated group. Elevated interstitial fibrosis scores in the SL-treated groups were decreased in both 7- and 14-day NES-1 treated groups, while alpha-smooth muscle actin (α-SMA) and apoptosis scores were depressed in both NES-1 treated groups. CONCLUSION: The present data demonstrate that UUO-induced renal fibrosis is ameliorated by NES-1, which appears to involve the inhibition of neutrophil infiltration and thereby amelioration of oxidative stress and inflammation. These data suggest that NES-1 may have a regulatory role in protecting the kidneys against obstruction-induced renal injury.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Obstrucción Ureteral / Enfermedades Renales Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Animals / Humans / Male Idioma: En Revista: Nephrol Dial Transplant Asunto de la revista: NEFROLOGIA / TRANSPLANTE Año: 2022 Tipo del documento: Article País de afiliación: Turquía Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Obstrucción Ureteral / Enfermedades Renales Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Animals / Humans / Male Idioma: En Revista: Nephrol Dial Transplant Asunto de la revista: NEFROLOGIA / TRANSPLANTE Año: 2022 Tipo del documento: Article País de afiliación: Turquía Pais de publicación: Reino Unido