Indole-3-propionic acid attenuates high glucose induced ER stress response and augments mitochondrial function by modulating PERK-IRE1-ATF4-CHOP signalling in experimental diabetic neuropathy.

Gundu, Chayanika; Arruri, Vijay Kumar; Sherkhane, Bhoomika; Khatri, Dharmendra Kumar; Singh, Shashi Bala

Gundu, Chayanika; Arruri, Vijay Kumar; Sherkhane, Bhoomika; Khatri, Dharmendra Kumar; Singh, Shashi Bala.

Afiliación

Gundu C; Molecular and Cellular Neuroscience Laboratory, Department of Pharmacology and Toxicology, National Institute of Pharmaceutical Education and Research (NIPER)-Hyderabad, Balanagar, India.
Arruri VK; Department of Neurosurgery, University of Wisconsin-Madison, Madison, WI, USA.
Sherkhane B; Molecular and Cellular Neuroscience Laboratory, Department of Pharmacology and Toxicology, National Institute of Pharmaceutical Education and Research (NIPER)-Hyderabad, Balanagar, India.
Khatri DK; Molecular and Cellular Neuroscience Laboratory, Department of Pharmacology and Toxicology, National Institute of Pharmaceutical Education and Research (NIPER)-Hyderabad, Balanagar, India.
Singh SB; Molecular and Cellular Neuroscience Laboratory, Department of Pharmacology and Toxicology, National Institute of Pharmaceutical Education and Research (NIPER)-Hyderabad, Balanagar, India.

Arch Physiol Biochem ; : 1-14, 2022 Jan 11.

Article en En | MEDLINE | ID: mdl-35015592

Palabras clave

ER stress; Indole-3-propionic acid; diabetic neuropathy; mitochondrial dysfunction; neuro2a cells

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Arch Physiol Biochem Asunto de la revista: BIOQUIMICA / FISIOLOGIA Año: 2022 Tipo del documento: Article País de afiliación: India Pais de publicación: Reino Unido

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