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Neochlorogenic Acid Attenuates Hepatic Lipid Accumulation and Inflammation via Regulating miR-34a In Vitro.
Yu, Meng-Hsun; Hung, Tung-Wei; Wang, Chi-Chih; Wu, Sheng-Wen; Yang, Tzu-Wei; Yang, Ching-Yu; Tseng, Tsui-Hwa; Wang, Chau-Jong.
Afiliación
  • Yu MH; Institute of Medicine, Chung Shan Medical University, Taichung 40201, Taiwan.
  • Hung TW; Department of Health Industry Technology Management, Chung Shan Medical University, Taichung 40201, Taiwan.
  • Wang CC; Department of Medicine, Division of Nephrology, Chung Shan Medical University Hospital, Taichung 40201, Taiwan.
  • Wu SW; School of Medicine, Chung Shan Medical University, Taichung 40201, Taiwan.
  • Yang TW; School of Medicine, Chung Shan Medical University, Taichung 40201, Taiwan.
  • Yang CY; Division of Gastroenterology and Hepatology, Department of Internal Medicine, Chung Shan Medical University Hospital, Taichung 40201, Taiwan.
  • Tseng TH; Department of Medicine, Division of Nephrology, Chung Shan Medical University Hospital, Taichung 40201, Taiwan.
  • Wang CJ; School of Medicine, Chung Shan Medical University, Taichung 40201, Taiwan.
Int J Mol Sci ; 22(23)2021 Dec 06.
Article en En | MEDLINE | ID: mdl-34884968
Neochlorogenic acid (5-Caffeoylquinic acid; 5-CQA), a major phenolic compound isolated from mulberry leaves, possesses anti-oxidative and anti-inflammatory effects. Although it modulates lipid metabolism, the molecular mechanism is unknown. Using an in-vitro model of nonalcoholic fatty liver disease (NAFLD) in which oleic acid (OA) induced lipid accumulation in HepG2 cells, we evaluated the alleviation effect of 5-CQA. We observed that 5-CQA improved OA-induced intracellular lipid accumulation by downregulating sterol regulatory element-binding protein 1 (SREBP1) and fatty acid synthase (FASN) expression, which regulates the fatty acid synthesis, as well as SREBP2 and HMG-CoA reductases (HMG-CoR) expressions, which regulate cholesterol synthesis. Treatment with 5-CQA also increased the expression of fatty acid ß-oxidation enzymes. Remarkably, 5-CQA attenuated OA-induced miR-34a expression. A transfection assay with an miR-34a mimic or miR-34a inhibitor revealed that miR-34a suppressed Moreover, Sirtuin 1 (SIRT1) expression and inactivated 5' adenosine monophosphate-activated protein kinase (AMPK). Our results suggest that 5-CQA alleviates lipid accumulation by downregulating miR-34a, leading to activation of the SIRT1/AMPK pathway.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ácido Quínico / Ácido Clorogénico / MicroARNs / Lipogénesis / Proteínas Quinasas Activadas por AMP / Sirtuina 1 / Inflamación / Hígado Tipo de estudio: Etiology_studies Límite: Humans Idioma: En Revista: Int J Mol Sci Año: 2021 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Ácido Quínico / Ácido Clorogénico / MicroARNs / Lipogénesis / Proteínas Quinasas Activadas por AMP / Sirtuina 1 / Inflamación / Hígado Tipo de estudio: Etiology_studies Límite: Humans Idioma: En Revista: Int J Mol Sci Año: 2021 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Suiza