Your browser doesn't support javascript.
loading
A primer on harnessing non-enzymatic post-translational modifications for drug design.
Long, Marcus J C; Ly, Phillippe; Aye, Yimon.
Afiliación
  • Long MJC; University of Lausanne 1015 Lausanne Switzerland.
  • Ly P; Swiss Federal Institute of Technology in Lausanne (EPFL) 1015 Lausanne Switzerland yimon.aye@epfl.ch.
  • Aye Y; Swiss Federal Institute of Technology in Lausanne (EPFL) 1015 Lausanne Switzerland yimon.aye@epfl.ch.
RSC Med Chem ; 12(11): 1797-1807, 2021 Nov 17.
Article en En | MEDLINE | ID: mdl-34825181
Of the manifold concepts in drug discovery and design, covalent drugs have re-emerged as one of the most promising over the past 20-or so years. All such drugs harness the ability of a covalent bond to drive an interaction between a target biomolecule, typically a protein, and a small molecule. Formation of a covalent bond necessarily prolongs target engagement, opening avenues to targeting shallower binding sites, protein complexes, and other difficult to drug manifolds, amongst other virtues. This opinion piece discusses frameworks around which to develop covalent drugs. Our argument, based on results from our research program on natural electrophile signaling, is that targeting specific residues innately involved in native signaling programs are ideally poised to be targeted by covalent drugs. We outline ways to identify electrophile-sensing residues, and discuss how studying ramifications of innate signaling by endogenous molecules can provide a means to predict drug mechanism and function and assess on- versus off-target behaviors.

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: RSC Med Chem Año: 2021 Tipo del documento: Article Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: RSC Med Chem Año: 2021 Tipo del documento: Article Pais de publicación: Reino Unido