Your browser doesn't support javascript.
loading
Hexafluoropropylene oxide dimer acid (GenX) exposure induces apoptosis in HepG2 cells.
Yoo, Hee Joon; Pyo, Min Cheol; Park, Yoonjin; Kim, Bo Yong; Lee, Kwang-Won.
Afiliación
  • Yoo HJ; Department of Biotechnology, College of Life Science and Biotechnology, Korea University, Seoul 02841, Republic of Korea.
  • Pyo MC; Department of Biotechnology, College of Life Science and Biotechnology, Korea University, Seoul 02841, Republic of Korea.
  • Park Y; Department of Clinical Laboratory Sciences, College of Health Science, Korea University, Seoul 02841, Republic of Korea.
  • Kim BY; Department of Clinical Laboratory Sciences, College of Health Science, Korea University, Seoul 02841, Republic of Korea.
  • Lee KW; Department of Biotechnology, College of Life Science and Biotechnology, Korea University, Seoul 02841, Republic of Korea.
Heliyon ; 7(11): e08272, 2021 Nov.
Article en En | MEDLINE | ID: mdl-34765786
Hexafluoropropylene oxide dimer acid, also known as GenX, is a poly- and perfluoroalkyl substance (PFAS). PFASs are nonvolatile synthetic substances that can be readily disseminated into the environment during processing and use, making them easy to implement in the soil, drinking water, and air. Compared to other PFASs, GenX has a comparatively short carbon chain length and is expected to have a lower tendency to accumulate in humans; therefore, GenX has recently been used as a substitute to other PFASs. However, the mechanisms underlying GenX action and intoxication in humans remains unclear. In this study, the apoptotic capacity of GenX in human liver cells was investigated. When representative human-derived liver cells (HepG2 cells) were treated with GenX for 12 h, cell viability was reduced, and apoptosis was greatly increased. In addition, GenX increased the generation of intracellular reactive oxygen species (ROS), indicating the induction of oxidative stress in a dose-dependent manner. GenX treatment increased the expression of major apoptosis-related genes relative to the untreated control group. This research indicates that GenX causes apoptosis through ROS mediation in HepG2 cells, which may expand our knowledge of the molecular and toxicological mechanisms of GenX.
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Heliyon Año: 2021 Tipo del documento: Article Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Heliyon Año: 2021 Tipo del documento: Article Pais de publicación: Reino Unido