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The CRL4DCAF1 cullin-RING ubiquitin ligase is activated following a switch in oligomerization state.
Mohamed, Weaam I; Schenk, Andreas D; Kempf, Georg; Cavadini, Simone; Basters, Anja; Potenza, Alessandro; Abdul Rahman, Wassim; Rabl, Julius; Reichermeier, Kurt; Thomä, Nicolas H.
Afiliación
  • Mohamed WI; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Schenk AD; University of Basel, Basel, Switzerland.
  • Kempf G; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Cavadini S; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Basters A; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Potenza A; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Abdul Rahman W; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Rabl J; University of Basel, Basel, Switzerland.
  • Reichermeier K; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
  • Thomä NH; Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
EMBO J ; 40(22): e108008, 2021 11 15.
Article en En | MEDLINE | ID: mdl-34595758
The cullin-4-based RING-type (CRL4) family of E3 ubiquitin ligases functions together with dedicated substrate receptors. Out of the ˜29 CRL4 substrate receptors reported, the DDB1- and CUL4-associated factor 1 (DCAF1) is essential for cellular survival and growth, and its deregulation has been implicated in tumorigenesis. We carried out biochemical and structural studies to examine the structure and mechanism of the CRL4DCAF1 ligase. In the 8.4 Å cryo-EM map of CRL4DCAF1 , four CUL4-RBX1-DDB1-DCAF1 protomers are organized into two dimeric sub-assemblies. In this arrangement, the WD40 domain of DCAF1 mediates binding with the cullin C-terminal domain (CTD) and the RBX1 subunit of a neighboring CRL4DCAF1 protomer. This renders RBX1, the catalytic subunit of the ligase, inaccessible to the E2 ubiquitin-conjugating enzymes. Upon CRL4DCAF1 activation by neddylation, the interaction between the cullin CTD and the neighboring DCAF1 protomer is broken, and the complex assumes an active dimeric conformation. Accordingly, a tetramerization-deficient CRL4DCAF1 mutant has higher ubiquitin ligase activity compared to the wild-type. This study identifies a novel mechanism by which unneddylated and substrate-free CUL4 ligases can be maintained in an inactive state.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Serina-Treonina Quinasas / Ubiquitina-Proteína Ligasas Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: EMBO J Año: 2021 Tipo del documento: Article País de afiliación: Suiza Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Serina-Treonina Quinasas / Ubiquitina-Proteína Ligasas Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: EMBO J Año: 2021 Tipo del documento: Article País de afiliación: Suiza Pais de publicación: Reino Unido