Your browser doesn't support javascript.
loading
Induction of IL-22-Producing CD4+ T Cells by Segmented Filamentous Bacteria Independent of Classical Th17 Cells.
Roy, Urmi; de Oliveira, Rômulo S; Galvez, Eric J C; Gronow, Achim; Basic, Marijana; Perez, Laura Garcia; Gagliani, Nicola; Bleich, Andre; Huber, Samuel; Strowig, Till.
Afiliación
  • Roy U; Department of Microbial Immune Regulation, Helmholtz Centre for Infection Research, Braunschweig, Germany.
  • de Oliveira RS; Department of Microbial Immune Regulation, Helmholtz Centre for Infection Research, Braunschweig, Germany.
  • Galvez EJC; Department of Microbial Immune Regulation, Helmholtz Centre for Infection Research, Braunschweig, Germany.
  • Gronow A; Department of Microbial Immune Regulation, Helmholtz Centre for Infection Research, Braunschweig, Germany.
  • Basic M; Institute for Laboratory Animal Science, Hannover Medical School, Hannover, Germany.
  • Perez LG; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Gagliani N; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Bleich A; Department of General, Visceral and Thoracic Surgery, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Huber S; Institute for Laboratory Animal Science, Hannover Medical School, Hannover, Germany.
  • Strowig T; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
Front Immunol ; 12: 671331, 2021.
Article en En | MEDLINE | ID: mdl-34566952
The intestinal microbiota modulates IL-22 production in the intestine, including the induction of IL-22-producing CD4+ T helper cells. Which specific bacteria are responsible for the induction of these cells is less well understood. Here, we demonstrate through the use of novel gnotobiotic knock-in reporter mice that segmented filamentous bacteria (SFB), which are known for their ability to induce Th17 cells, also induce distinct IL-17A negative CD4+ T cell populations in the intestine. A subset of these cells instead produces IL-22 upon restimulation ex vivo and also during enteric infections. Furthermore, they produce a distinct set of cytokines compared to Th17 cells including the differential expression of IL-17F and IFN-γ. Importantly, genetic models demonstrate that these cells, presumably Th22 cells, develop independently of intestinal Th17 cells. Together, our data identifies that besides Th17, SFB also induces CD4+ T cell populations, which serve as immediate source of IL-22 during intestinal inflammation.
Asunto(s)
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos T CD4-Positivos / Interleucinas / Células Th17 / Microbioma Gastrointestinal Límite: Animals Idioma: En Revista: Front Immunol Año: 2021 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Linfocitos T CD4-Positivos / Interleucinas / Células Th17 / Microbioma Gastrointestinal Límite: Animals Idioma: En Revista: Front Immunol Año: 2021 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Suiza