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Elucidating the Mechanisms Underlying the Signal Drift of Electrochemical Aptamer-Based Sensors in Whole Blood.
Leung, Kaylyn K; Downs, Alex M; Ortega, Gabriel; Kurnik, Martin; Plaxco, Kevin W.
Afiliación
  • Leung KK; Department of Chemistry and Biochemistry, University of California Santa Barbara, Santa Barbara, California 93106, United States.
  • Downs AM; Center for Bioengineering, University of California Santa Barbara, Santa Barbara, California 93106, United States.
  • Ortega G; Center for Bioengineering, University of California Santa Barbara, Santa Barbara, California 93106, United States.
  • Kurnik M; Department of Mechanical Engineering, University of California Santa Barbara, Santa Barbara, California 93106, United States.
  • Plaxco KW; Department of Chemistry and Biochemistry, University of California Santa Barbara, Santa Barbara, California 93106, United States.
ACS Sens ; 6(9): 3340-3347, 2021 09 24.
Article en En | MEDLINE | ID: mdl-34491055
The ability to monitor drugs, metabolites, hormones, and other biomarkers in situ in the body would greatly advance both clinical practice and biomedical research. To this end, we are developing electrochemical aptamer-based (EAB) sensors, a platform technology able to perform real-time, in vivo monitoring of specific molecules irrespective of their chemical or enzymatic reactivity. An important obstacle to the deployment of EAB sensors in the challenging environments found in the living body is signal drift, whereby the sensor signal decreases over time. To date, we have demonstrated a number of approaches by which this drift can be corrected sufficiently well to achieve good measurement precision over multihour in vivo deployments. To achieve a much longer in vivo measurement duration, however, will likely require that we understand and address the sources of this effect. In response, here, we have systematically examined the mechanisms underlying the drift seen when EAB sensors and simpler, EAB-like devices are challenged in vitro at 37 °C in whole blood as a proxy for in vivo conditions. Our results demonstrate that electrochemically driven desorption of a self-assembled monolayer and fouling by blood components are the two primary sources of signal loss under these conditions, suggesting targeted approaches to remediating this degradation and thus improving the stability of EAB sensors and other, similar electrochemical biosensor technologies when deployed in the body.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: ACS Sens Año: 2021 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: ACS Sens Año: 2021 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos