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Genotyping of interleukins-18 promoters and their correlation with coronary artery stenosis in Saudi population.
Tabrez, Shams; Jabir, Nasimudeen R; Zughaibi, Torki A; Shakil, Shazi.
Afiliación
  • Tabrez S; King Fahd Medical Research Center, King Abdulaziz University, P.O. Box 80216, Jeddah, 21589, Saudi Arabia. shamstabrez1@gmail.com.
  • Jabir NR; Department of Medical Laboratory Technology, Faculty of Applied Medical Sciences, King Abdulaziz University, P.O. Box 80216, Jeddah, 21589, Saudi Arabia. shamstabrez1@gmail.com.
  • Zughaibi TA; Department of Biochemistry, Centre for Research and Development, PRIST University, Tamil Nadu, Vallam, Thanjavur, 613403, India.
  • Shakil S; King Fahd Medical Research Center, King Abdulaziz University, P.O. Box 80216, Jeddah, 21589, Saudi Arabia.
Mol Biol Rep ; 48(10): 6695-6702, 2021 Oct.
Article en En | MEDLINE | ID: mdl-34431037
BACKGROUND: Complex coronary atherosclerotic lesions often lead to coronary occlusion, clinically represented as a single-vessel disease (SVD) and multivessel disease (MVD). These occlusions could hinder the blood flow in coronary arteries that affects appropriate management of the CVD. The current study intended to genotype interleukin (IL)-18 promoter's hotspots (rs187238, rs1946518, and rs1946519) and their possible association with coronary artery stenosis. MATERIAL AND METHODS: The IL-18 promoter genotyping was performed by the Sanger method along with the examination of biochemical parameters in 125 study subjects categorized into three groups, viz. controls, SVD and MVD. RESULTS: The current study observed a significant association of diabetes, hypertension, and dyslipidemia between the studied group's viz. healthy controls, SVD, and MVD. Fasting blood sugar and glycosylated hemoglobin (HBA1C) were also significantly enhanced from 4.82 vs. 8.01 and 4.33 vs. 8.27, in SVD, and MVD respectively. Despite the visible differences in the pattern of genotypic and allelic expressions, the current study did not show any statistically significant correlation with IL-18 promoter polymorphism at its hotspots with controls, SVD, and MVD subjects. The only exception of the above results was the distribution of allelic frequency at the rs1946519 hotspot, where a significant change (P < 0.05) was observed. CONCLUSION: This study is of additional value to our previous reports, revealing the pattern of genotypes and allelic frequency of IL-18 promoters in a small cohort of Saudi ethnicity. Further investigations on larger sample size are recommended to envisage the presence of functional mutations in the IL-18 gene that could establish or rule out the possible association of IL-18 polymorphism with SVD and MVD.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Regiones Promotoras Genéticas / Predisposición Genética a la Enfermedad / Interleucina-18 / Estenosis Coronaria / Técnicas de Genotipaje Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Female / Humans / Male / Middle aged País/Región como asunto: Asia Idioma: En Revista: Mol Biol Rep Año: 2021 Tipo del documento: Article País de afiliación: Arabia Saudita Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Regiones Promotoras Genéticas / Predisposición Genética a la Enfermedad / Interleucina-18 / Estenosis Coronaria / Técnicas de Genotipaje Tipo de estudio: Observational_studies / Prognostic_studies / Risk_factors_studies Límite: Female / Humans / Male / Middle aged País/Región como asunto: Asia Idioma: En Revista: Mol Biol Rep Año: 2021 Tipo del documento: Article País de afiliación: Arabia Saudita Pais de publicación: Países Bajos