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Dihydrosphingosine driven enrichment of sphingolipids attenuates TGFß induced collagen synthesis in cardiac fibroblasts.
Magaye, Ruth R; Savira, Feby; Xiong, Xin; Huynh, Kevin; Meikle, Peter J; Reid, Christopher; Flynn, Bernard L; Kaye, David; Liew, Danny; Wang, Bing H.
Afiliación
  • Magaye RR; Biomarker Discovery Laboratory, Baker Heart and Diabetes Institute, Melbourne, Australia.
  • Savira F; Monash Centre of Cardiovascular Research and Education in Therapeutics, Melbourne, Australia.
  • Xiong X; Biomarker Discovery Laboratory, Baker Heart and Diabetes Institute, Melbourne, Australia.
  • Huynh K; Monash Centre of Cardiovascular Research and Education in Therapeutics, Melbourne, Australia.
  • Meikle PJ; Biomarker Discovery Laboratory, Baker Heart and Diabetes Institute, Melbourne, Australia.
  • Reid C; Monash Centre of Cardiovascular Research and Education in Therapeutics, Melbourne, Australia.
  • Flynn BL; Shanghai Institute of Heart Failure, Research Centre for Translational Medicine, Shanghai East Hospital, Tongji University, School of Medicine, Shanghai 200120, PR China.
  • Kaye D; Metabolomics Research Group, Baker Heart and Diabetes Institute, Melbourne, Australia.
  • Liew D; Metabolomics Research Group, Baker Heart and Diabetes Institute, Melbourne, Australia.
  • Wang BH; School of Public Health School, Curtin University, Perth, Australia.
Int J Cardiol Heart Vasc ; 35: 100837, 2021 Aug.
Article en En | MEDLINE | ID: mdl-34277924
The sphingolipid de novo synthesis pathway, encompassing the sphingolipids, the enzymes and the cell membrane receptors, are being investigated for their role in diseases and as potential therapeutic targets. The intermediate sphingolipids such as dihydrosphingosine (dhSph) and sphingosine (Sph) have not been investigated due to them being thought of as precursors to other more active lipids such as ceramide (Cer) and sphingosine 1 phosphate (S1P). Here we investigated their effects in terms of collagen synthesis in primary rat neonatal cardiac fibroblasts (NCFs). Our results in NCFs showed that both dhSph and Sph did not induce collagen synthesis, whilst dhSph reduced collagen synthesis induced by transforming growth factor ß (TGFß). The mechanisms of these inhibitory effects were associated with the increased activation of the de novo synthesis pathway that led to increased dihydrosphingosine 1 phosphate (dhS1P). Subsequently, through a negative feedback mechanism that may involve substrate-enzyme receptor interactions, S1P receptor 1 expression (S1PR1) was reduced.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Int J Cardiol Heart Vasc Año: 2021 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Irlanda

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Int J Cardiol Heart Vasc Año: 2021 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Irlanda