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Proton Transport in Cancer Cells: The Role of Carbonic Anhydrases.
Becker, Holger M; Deitmer, Joachim W.
Afiliación
  • Becker HM; Zoology and Animal Physiology, Institute of Zoology, TU Dresden, D-01217 Dresden, Germany.
  • Deitmer JW; Department of Biology, University of Kaiserslautern, D-67653 Kaiserslautern, Germany.
Int J Mol Sci ; 22(6)2021 Mar 20.
Article en En | MEDLINE | ID: mdl-33804674
Intra- and extracellular pH regulation is a pivotal function of all cells and tissues. Net outward transport of H+ is a prerequisite for normal physiological function, since a number of intracellular processes, such as metabolism and energy supply, produce acid. In tumor tissues, distorted pH regulation results in extracellular acidification and the formation of a hostile environment in which cancer cells can outcompete healthy local host cells. Cancer cells employ a variety of H+/HCO3--coupled transporters in combination with intra- and extracellular carbonic anhydrase (CA) isoforms, to alter intra- and extracellular pH to values that promote tumor progression. Many of the transporters could closely associate to CAs, to form a protein complex coined "transport metabolon". While transport metabolons built with HCO3--coupled transporters require CA catalytic activity, transport metabolons with monocarboxylate transporters (MCTs) operate independently from CA catalytic function. In this article, we assess some of the processes and functions of CAs for tumor pH regulation and discuss the role of intra- and extracellular pH regulation for cancer pathogenesis and therapeutic intervention.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Protones / Anhidrasas Carbónicas / Neoplasias Tipo de estudio: Etiology_studies Límite: Animals / Humans Idioma: En Revista: Int J Mol Sci Año: 2021 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Suiza

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Protones / Anhidrasas Carbónicas / Neoplasias Tipo de estudio: Etiology_studies Límite: Animals / Humans Idioma: En Revista: Int J Mol Sci Año: 2021 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Suiza