Your browser doesn't support javascript.
loading
Negative regulation of plastidial isoprenoid pathway by herbivore-induced ß-cyclocitral in Arabidopsis thaliana.
Mitra, Sirsha; Estrada-Tejedor, Roger; Volke, Daniel C; Phillips, Michael A; Gershenzon, Jonathan; Wright, Louwrance P.
Afiliación
  • Mitra S; Department of Biochemistry, Max Planck Institute for Chemical Ecology, 07745 Jena, Germany; smitra@unipune.ac.in lwright@tutanota.com.
  • Estrada-Tejedor R; Department of Botany, Savitribai Phule Pune University, Pune-411007, India.
  • Volke DC; Pharmaceutical Chemistry Group, IQS School of Engineering, Universitat Ramon Llull, 08017 Barcelona, Spain.
  • Phillips MA; The Novo Nordisk Foundation Center for Biosustainability, Technical University of Denmark, 2800 Kongens Lyngby, Denmark.
  • Gershenzon J; Department of Biology, University of Toronto Mississauga, Mississauga, ON L5L 1C6, Canada.
  • Wright LP; Department of Biochemistry, Max Planck Institute for Chemical Ecology, 07745 Jena, Germany.
Proc Natl Acad Sci U S A ; 118(10)2021 03 09.
Article en En | MEDLINE | ID: mdl-33674379
Insect damage to plants is known to up-regulate defense and down-regulate growth processes. While there are frequent reports about up-regulation of defense signaling and production of defense metabolites in response to herbivory, much less is understood about the mechanisms by which growth and carbon assimilation are down-regulated. Here we demonstrate that insect herbivory down-regulates the 2-C-methyl-D-erythritol-4-phosphate (MEP) pathway in Arabidopsis (Arabidopsis thaliana), a pathway making primarily metabolites for use in photosynthesis. Simulated feeding by the generalist herbivore Spodoptera littoralis suppressed flux through the MEP pathway and decreased steady-state levels of the intermediate 1-deoxy-D-xylulose 5-phosphate (DXP). Simulated herbivory also increased reactive oxygen species content which caused the conversion of ß-carotene to ß-cyclocitral (ßCC). This volatile oxidation product affected the MEP pathway by directly inhibiting DXP synthase (DXS), the rate-controlling enzyme of the MEP pathway in Arabidopsis and inducing plant resistance against S. littoralis ßCC inhibited both DXS transcript accumulation and DXS activity. Molecular models suggested that ßCC binds to DXS at the binding site for the thymine pyrophosphate cofactor and blocks catalysis, which was confirmed by direct assays of ßCC with the purified DXS protein in vitro. Another intermediate of the MEP pathway, 2-C-methyl-D-erythritol-2, 4-cyclodiphosphate, which is known to stimulate salicylate defense signaling, showed greater accumulation and enhanced export out of the plastid in response to simulated herbivory. Together, our work implicates ßCC as a signal of herbivore damage in Arabidopsis that increases defense and decreases flux through the MEP pathway, a pathway involved in growth and carbon assimilation.
Asunto(s)
Palabras clave

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Terpenos / Arabidopsis / Plastidios / Diterpenos / Aldehídos Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2021 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Terpenos / Arabidopsis / Plastidios / Diterpenos / Aldehídos Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2021 Tipo del documento: Article Pais de publicación: Estados Unidos