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Homeostatic p62 levels and inclusion body formation in CHCHD2 knockout mice.
Sato, Shigeto; Noda, Sachiko; Torii, Satoru; Amo, Taku; Ikeda, Aya; Funayama, Manabu; Yamaguchi, Junji; Fukuda, Takahiro; Kondo, Hiromi; Tada, Norihiro; Arakawa, Satoko; Watanabe, Masahiko; Uchiyama, Yasuo; Shimizu, Shigeomi; Hattori, Nobutaka.
Afiliación
  • Sato S; Department of Neurology, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Noda S; Department of Neurology, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Torii S; Department of Pathological Cell Biology, Medical Research Institute, Tokyo Medical and Dental University, Tokyo 113-8510, Japan.
  • Amo T; Department of Applied Chemistry, National Defense Academy, Yokosuka 239-8686, Japan.
  • Ikeda A; Department of Neurology, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Funayama M; Research Institute for Diseases of Old Age, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
  • Yamaguchi J; Department of Cellular and Molecular Neuropathology, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Fukuda T; Laboratory of Morphology and Image Analysis, Research Support Center, Juntendo University Graduate School of Medicine, 113-8421, Japan.
  • Kondo H; Division of Neuropathology, Department of Neuropathology, The Jikei University School of Medicine, Tokyo 105-8461, Japan.
  • Tada N; Histology Center, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
  • Arakawa S; Atopy Research Center, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Watanabe M; Department of Pathological Cell Biology, Medical Research Institute, Tokyo Medical and Dental University, Tokyo 113-8510, Japan.
  • Uchiyama Y; Department of Anatomy, Faculty of Medicine, Hokkaido University, Sapporo 060-8638, Japan.
  • Shimizu S; Department of Cellular and Molecular Neuropathology, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Hattori N; Department of Pathological Cell Biology, Medical Research Institute, Tokyo Medical and Dental University, Tokyo 113-8510, Japan.
Hum Mol Genet ; 30(6): 443-453, 2021 04 30.
Article en En | MEDLINE | ID: mdl-33631794
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Factores de Transcripción / Cuerpos de Inclusión / Cuerpos de Lewy / Proteínas de Unión al ADN / Proteína Sequestosoma-1 / Homeostasis / Mitocondrias Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2021 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Reino Unido
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Factores de Transcripción / Cuerpos de Inclusión / Cuerpos de Lewy / Proteínas de Unión al ADN / Proteína Sequestosoma-1 / Homeostasis / Mitocondrias Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2021 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Reino Unido