AMPK-mTOR-ULK1-mediated autophagy protects carbon tetrachloride-induced acute hepatic failure by inhibiting p21 in rats.

Wang, Qiwen; Liu, Weixia; Liu, Gaopeng; Li, Pan; Guo, Xueqiang; Zhang, Chunyan

Wang, Qiwen; Liu, Weixia; Liu, Gaopeng; Li, Pan; Guo, Xueqiang; Zhang, Chunyan.

Afiliación

Wang Q; State Key Laboratory Cell Differentiation and Regulation, Henan Normal University, #46 East of Construction Road, Xinxiang, 453007 Henan, China.
Liu W; Henan International Joint Laboratory of Pulmonary Fibrosis, Henan Normal University, #46 East of Construction Road, Xinxiang, 453007 Henan, China.
Liu G; Henan Center for Outstanding Overseas Scientists of Pulmonary Fibrosis, Henan Normal University, #46 East of Construction Road, Xinxiang, 453007 Henan, China.
Li P; College of Life Science, Henan Normal University, #46 East of Construction Road, Xinxiang, 453007 Henan, China.
Guo X; Institute of Biomedical Science, Henan Normal University, #46 East of Construction Road, Xinxiang, 453007 Henan, China.
Zhang C; Overseas Expertise Introduction Center for Discipline Innovation of Pulmonary Fibrosis (111 Project), Henan Normal University, #46 East of Construction Road, Xinxiang, 453007 Henan, China.

J Toxicol Pathol ; 34(1): 73-82, 2021 Jan.

Article en En | MEDLINE | ID: mdl-33627946

Palabras clave

AMPK-mTOR-ULK1 axis; autophagy; carbon tetrachloride (CCl4); p21; rat

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: J Toxicol Pathol Año: 2021 Tipo del documento: Article País de afiliación: China Pais de publicación: Japón

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